Despite the reliability and high success rates related to treatment with implants, failures of implant-supported oral rehabilitation are still persistent. These failures can cause both mechanical damage, such as loosening the retaining screw connections due to instability, as biological damage, such as inflammatory reactions in periimplants tissues due to the occurrence of bacterial leakage along the implant/abutment interface and the subsequent colonization of within the implant and its components. The mechanisms responsible for these mechanical/biological failures of implant systems are not fully elucidated, and changes to the models of prosthetic connections and retaining screws have been developed in order to increase the stability of connections and solve these problems. However, the literature on the ideal of these changes and their determinants involved setting is inconclusive. The objective of this study is to microbiologically evaluate by the DNA Checkerboard hybridization technique, the occurrence of bacterial infiltration from the external environment to the interior of the implant through these different implant/abutment interface in two different implant systems, external hexagonal (HE) and internal triangle (TI), with conventional plane screws and experimental tapered screws of abutments retention.
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