Marcela Sorelli Carneiro Ramos
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Ministério da Educação (Brasil). Universidade Federal do ABC (UFABC). Centro de Ciências Naturais e Humanas (CCNH) (Institutional affiliation for the last research proposal) Birthplace: Brazil
Marcela Sorelli Carneiro Ramos in research grants and scholarships supported by FAPESP.
- TLR4 and complement system : possible key mechanism in renal ischemia/reperfusion induced cardiac hypertrophy, AP.R
Abstract
The immune system interacts with several different tissues and organs of the human body, for example, the heart tissue. In diseases that generate a systemic inflammation, some factors are released in the blood stream and they are able to reach the most diverse organs, promoting interaction between tissue cells and immunity cells. It is known that renal insufficiency (RI) is characterize...
- Impact of inflammatory response induced by acute renal ischemia in cardiac tissue, AP.JP
Abstract
Cardiac remodeling occurs primarily as an adaptive response to an increase in demand, commonly resulting in the so called cardiac hypertrophy. The heart is a target organ in many regulatory systems, such as hormones, neuronal and hemodynamic factors, which modulate heart trophism direct or indirectly. Besides these actions, largely described in the literature, it is well known the heart...
- Thyroid hormone increases beta-catenin levels through PI3K in rat heart in vivo and in vitro, AR.EXT
- Cardiac alterations induced by ischemic renal injury using experimental models in vitro: participation of nitric oxide, BP.IC
Abstract
Cardiovascular diseases (CVD) are the cause of much of the deaths, due not only to genetic factors, but also to environmental factors, due to the modern way of life. However, they may be secondary to other diseases, such as cardiorenal syndromes (SCRs). Cardiorenal syndromes are diseases that show the intense connection between the functioning of the kidneys and the heart and are subdiv...
- Role of the Klotho/FGF23 axis in the development of Type 3 Cardiorenal Syndrome induced by ischemic renal injury, BP.DD
Abstract
In diseases that generate an inflammatory situation, such as Cardiorenal Syndrome, different clinical conditions promote cardiac and renal dysfunction. It has five different types and the first two (types 1 and 2) are associated anormalities in cardiac function that cause injury or renal dysfunction; type 3 and 4 are characterized by renal insults that subject the heart to dysfunctions;...
- Contribution of TLR's/Inflammasome NLRP3/IL-1B axis in renal Ischemia/Reperfusion-induced electrophysiological disturbances, BP.PD
Abstract
Patients with chronic renal ischemia have a high risk to develop cardiovascular diseases. These diseases represent a 41% of death in patient in hemodialysis treatment. Therefore, understand the physiopathological mechanisms involved is critical. Recently, a study conducted by the proponent of the present project showed an elevated concentration of IL-1² 8 days after renal ischemia-rep...
- Participation of complement system in the cardiomyocyte hypertrophy induced by activation of toll-like receptor type 4 in vitro: the role of CaMKIId, BP.IC
Abstract
The immune system presents different interfaces among systems and other body tissues, such as heart tissue. In diseases that generate inflammatory condition, the factors released into the bloodstream can reach many more organs, promoting interaction between tissue cells and immune components. It is known that renal insufficiency (RI) is characterized by a systemic inflammatory conditio...
- Impact of the renal insufficiency in the in vivo cardiac tissue: contribution of cell apoptosis in cardiac remodeling, BP.IC
Abstract
The immune system presents different interrelations with other organs and tissues, such as the heart tissue. This system operates in several ways, particularly through the production and release of pro and anti inflammatory cytokines, which are capable of carrying on a range of biological effects related to cell proliferation and survival. One of the conditions responsible for the mass ...
(Only some records are available in English at this moment)
- Efects of the silencing and super-expressing of Klotho gene in cardiac function through hypertrophy induced by cardiorenal syndrome, BE.EP.DD
Abstract
The Cardiorenal Syndrome is characterized by the presence of a systemic inflammatory condition where different clinical conditions promote cardiac and renal dysfunction. It has five different types where the first two types (type 1 and 2) are associated with abnormalities in cardiac function that cause injury or renal dysfunction; those of type 3 and 4 that are characterized by renal in...
| 1 / 0 | Ongoing research grants |
| 4 / 3 | Completed research grants |
| 2 / 2 | Ongoing scholarships in Brazil |
| 9 / 5 | Completed scholarships in Brazil |
| 1 / 1 | Completed scholarships abroad |
| 17 / 11 | All research grants and scholarships |
| Associated processes | |
(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)
| Publications | 4 |
| Citations | 2 |
| Cit./Article | 0.5 |
| Data from Web of Science | |
TRENTIN-SONODA, MAYRA; FRATONI, FRAYLI MALTONI; DA CRUZ JUNHO, CAROLINA VICTORIA; SILVA, WELLINGTON CAIO; PANICO, KARINE; CARNEIRO-RAMOS, MARCELA SORELLI. Caspase-1 as Molecular Key in Cardiac Remodeling during Cardiorenal Syndrome Type 3 in the Murine Model. CURRENT MOLECULAR MEDICINE, v. 20, n. 1, p. 72-78, 2020. Web of Science Citations: 0. (15/15832-7, 15/19107-5)
CRUZ JUNHO, V, C.; TRENTIN-SONODA, M.; ALVIM, J. M.; GAISLER-SILVA, F.; CARNEIRO-RAMOS, M. S.. Ca(2+/)Calmodulin-dependent kinase II delta B is essential for cardiomyocyte hypertrophy and complement gene expression after LPS and HSP60 stimulation in vitro. Brazilian Journal of Medical and Biological Research, v. 52, n. 7, 2019. Web of Science Citations: 1. (15/01792-3, 14/16908-4, 16/11865-0, 15/19107-5)
CARNEIRO-RAMOS‚ MS; SILVA‚ VB; SANTOS‚ RAS; BARRETO-CHAVES‚ MLM. Tissue-specific modulation of angiotensin-converting enzyme (ACE) in hyperthyroidism. Peptides, v. 27, n. 11, p. 2942-2949, 2006.
LOPEZ ALARCON, MARIA MICAELA; TRENTIN-SONODA, MAYRA; PANICO, KARINE; SCHLEIER, YGOR; DUQUE, THABATA; MORENO-LOAIZA, OSCAR; DE YURRE, AINHOA RODRIGUEZ; FERREIRA, FABIANNO; CAIO-SILVA, WELLINGTON; COURY, PEDROSA ROBERTO; et al. Cardiac arrhythmias after renal I/R depend on IL-1 beta. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, v. 131, p. 101-111, JUN 2019. Web of Science Citations: 1. (08/10175-4, 17/05974-4)
CIRINO-SILVA, ROGERIO; KMIT, FERNANDA V.; TRENTIN-SONODA, MAYRA; NAKAMA, KARINA K.; PANICO, KARINE; ALVIM, JULIANA M.; DREYER, THIAGO R.; MARTINHO-SILVA, HERCULANO; CARNEIRO-RAMOS, MARCELA S.. Renal ischemia/reperfusion-induced cardiac hypertrophy in mice: Cardiac morphological and morphometric characterization. JRSM CARDIOVASCULAR DISEASE, v. 6, JAN 1 2017. Web of Science Citations: 0. (15/19107-5)
C.V. CRUZ JUNHO; M. TRENTIN-SONODA; J.M. ALVIM; F. GAISLER-SILVA; M.S. CARNEIRO-RAMOS. Ca2+/Calmodulin-dependent kinase II delta B is essential for cardiomyocyte hypertrophy and complement gene expression after LPS and HSP60 stimulation in vitro. Brazilian Journal of Medical and Biological Research, v. 52, n. 7, p. -, 2019. (16/11865-0, 15/01792-3, 14/16908-4, 15/19107-5)