CV Lattes
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Universidade de São Paulo (USP). Instituto de Ciências Biomédicas (ICB) (Institutional affiliation for the last research proposal) Birthplace: Brazil
Degree in Pharmacy and Biochemistry from University of São Paulo (USP), Master and PhD in Pharmacology from USP, and Post doctorate from the Massachusetts General Hospital - Harvard Medical School, Boston, USA. Associate-Professor at USP (2003) and Member of the São Paulo State Academy of Sciences (2012) and Head of Department of Pharmacology (2013-2017). He is currently Professor of Department of Pharmacology of the Institute of Biomedical Sciences at USP (2009-). Contributed in the field of Pharmacology and Neuroscience with emphasis on aging and signalling cascade linked to degenerative diseases by introducing new knowledge in studies of adaptive responses associated with hormesis and cognition involving cardiotonic steroids, glucocorticoids, Klotho protein, glutamate and intermittent fasting and . Member of Editorial Board of Plos ONe, British Journal Pharmacology, Frontiers in Endocrinology and Brazilian Journal of Medical and Biological Research. (Source: Lattes Curriculum)
The modulation of the glutamate activity via NMDA receptor has a strong influence in the adaptive processes that prevent premature aging, as well as play an important role in cognition and degenerative processes. It is well know that the insulin signaling pathways, mediators of the inflammatory response and Na,K-ATPase acting as enzyme and as a receiver of endogenous steroids (hormones wi…
Evidence has shown that OUA is release in a different time-course when compared to GC during a stress response. In addition, OUA also interferes with the CRH transport into the circulation indicating that this hormone plays an important role in the stress response. However, there is no data about the OUA effects during a chronic unpredictable stress. The present work aims to investigate t…
Systemic bacterial infections often result in enduring cognitive impairment and are a risk factor for dementia. There are currently no effective treatments for infection-induced cognitive impairment. Previous studies have shown that intermittent fasting (IF) can increase the resistance of neurons to injury and disease by stimulating adaptive cellular stress responses. However, the impact …
Studies have shown that dietary energy restriction (DER) without malnutrition can increase resistance of neurons to excitotoxic stress and may ameliorate age-related deficits in cognitive function through hormetic mechanisms involving activation of adaptive cellular stress response pathways. Fundamental to the hormetic adaptive response is gene expression regulation. Although different st…
(Only some records are available in English at this moment)
Alzheimer disease (AD) is one of the leading neurodegenerative pathologies in the western world and represents a health and socioeconomic problem. In this context , understanding the mechanisms underlying the pathogenesis of AD would be possible to identify new therapeutic options. The lost of the Endoplasmic Reticulum (ER) calcium homeostasis emerges as a central player in the developmen…
The highest prevalence of diseases related to aging like Parkinson's disease, Alzheimer's disease, amyotrophic lateral sclerosis and multiple sclerosis, are a result of the increase in life expectancy. Aging is a complex and multifactorial process in which molecular, cellular and behavior alterations can be observed. Inflammation is a common and important factor between many alterations o…
Aging is a complex and multifactorial process, associated with molecular, cellular and behavioral alterations. Oxidative stress, neuroinflammation and excitotoxicity are characteristics common to various neuropathological processes of aging as in Parkinson's disease, amyotrophic lateral sclerosis, multiple sclerosis and Alzheimer's disease. The neuronal damage by excitotoxicity occurs, ma…
GPNMB is a protein that is related to antiapoptotic mechanisms and recent studies suggest its neuroprotective role. GPNMB can be cleaved and its extracellular fragment interacts with a transmembrane protein called Na+/K+-ATPase (NKA). NKA is essential to electrolytic control of the cell and also has a signaling function that influences in inflammatory processes. Several studies suggest th…
Depression is an incapacitating and hardly treated psychiatric condition so the development of new drugs has been a priority for big pharmaceutical companies and academics. New molecular entities with antagonistic action on CRHR1 and V1b receptors have been developed by Sanofi-Aventis and Pfizer and showed antidepressant activities in pre-clinical models. We investigated the effectiveness…
(Only some records are available in English at this moment)
Alzheimer's disease (AD) is one of the leading neurodegenerative pathologies in the western world and represents a health and socioeconomic problem. In this context, understanding the mechanisms underlying the pathogenesis of AD would be possible to identify new therapeutic options. Neuroinflammation has an important role in AD. Microglia, the macrophages of the central nervous system pla…
The enzyme Na+,K+-ATPase (Sodium Potassium Adenosine Trisfosfatase) is an integral membrane ion pump, essential to maintain the osmotic balance of cells, by its energy-driven ability to export Na+ and import K+. Its functions are vital to all cells. Of the four ±1-4 subunit isoforms, ±1-3 are expressed in the central nervous system. The ±2 isoform is highly enriched in atrocytes and the ±…
Aging process was always investigated in C. elegans due to its reproductive cycle (they are hermaphrodites) and short lifespan (they live about 2 months). C. elegans life cycle can be indefinitely expanded once animals enter a phase known as dauer. Several genes altering this stage formation were identified and many of these genes codify proteins associated with the only tyrosine kinase r…
(Only some records are available in English at this moment)
| 1 / 1 | Ongoing research grants |
| 25 / 8 | Completed research grants |
| 2 / 2 | Ongoing scholarships in Brazil |
| 37 / 24 | Completed scholarships in Brazil |
| 7 / 6 | Completed scholarships abroad |
| 72 / 41 | All research grants and scholarships |
| Associated processes | |
(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)
| Publications | 28 |
| Citations | 283 |
| Cit./Article | 10.1 |
| Data from Web of Science | |
RAMOS, ADRIANA T.; TUFIK, SERGIO; TRONCONE, LANFRANCO R. P.. Control of Stress-Induced ACTH Secretion by Vasopressin and CRH: Additional Evidence. NEUROPSYCHOBIOLOGY, v. 73, n. 3, p. 184-190, 2016. Web of Science Citations: 2. (13/18897-7, 15/08098-5, 98/14303-3)
NOVAES, LEONARDO S.; DOS SANTOS, NILTON BARRETO; BATALHOTE, RAFAELA F. P.; MALTA, MARILIA BRINATI; CAMARINI, ROSANA; SCAVONE, CRISTOFORO; MUNHOZ, CAROLINA DEMARCHI. Environmental enrichment protects against stress-induced anxiety: Role of glucocorticoid receptor, ERK, and CREB signaling in the basolateral amygdala. Neuropharmacology, v. 113, n. A, p. 457-466, FEB 2017. Web of Science Citations: 22. (16/03572-3, 10/13843-8, 12/24727-4)
YSHII, LIDIA M.; GEBAUER, CHRISTINA M.; PIGNOLET, BEATRICE; MAURE, EMILIE; QUERIAULT, CLEMENCE; PIERAU, MANDY; SAITO, HIROMITSU; SUZUKI, NOBORU; BRUNNER-WEINZIERL, MONIKA; BAUER, JAN; et al. CTLA4 blockade elicits paraneoplastic neurological disease in a mouse model. BRAIN, v. 139, n. 11, p. 2923-2934, NOV 2016. Web of Science Citations: 13. (13/22196-4)
ORELLANA, ANA MARIA; LEITE, JACQUELINE ALVES; KINOSHITA, PAULA FERNANDA; VASCONCELOS, ANDREA RODRIGUES; ANDREOTTA, DIANA ZUKAS; LIMA, LARISSA DE SA; XAVIER, GILBERTO FERNANDO; KAWAMOTO, ELISA MITIKO; SCAVONE, CRISTOFORO. Ouabain increases neuronal branching in hippocampus and improves spatial memory. Neuropharmacology, v. 140, p. 260-274, SEP 15 2018. Web of Science Citations: 0. (16/07427-8, 11/22844-0)
VASCONCELOS, ANDREA RODRIGUES; DOS SANTOS, NILTON BARRETO; SCAVONE, CRISTOFORO; MUNHOZ, CAROLINA DEMARCHI. Nrf2/ARE Pathway Modulation by Dietary Energy Regulation in Neurological Disorders. FRONTIERS IN PHARMACOLOGY, v. 10, FEB 4 2019. Web of Science Citations: 4. (12/24727-4, 16/07427-8)
YSHII, LIDIA; GEBAUER, CHRISTINA; BERNARD-VALNET, RAPHAEL; LIBLAU, ROLAND. Neurons and Tcells: Understanding this interaction for inflammatory neurological diseases. European Journal of Immunology, v. 45, n. 10, p. 2712-2720, OCT 2015. Web of Science Citations: 12. (13/22196-4)
DE MELLO, NATALIA PRUDENTE; ANDREOTTI, DIANA ZUKAS; ORELLANA, ANA MARIA; SCAVONE, CRISTOFORO; KAWAMOTO, ELISA MITIKO. Inverse sex-based expression profiles of PTEN and Klotho in mice. SCIENTIFIC REPORTS, v. 10, n. 1, NOV 19 2020. Web of Science Citations: 0. (11/21308-8, 16/07427-8, 16/22996-9, 15/25491-2)
DE MOLLO, NATALIA PRUDENTE; ORELLANA, ANA MARIA; MAZUCANTI, CAIO HENRIQUE; LIMA, GEOVANNI DE MORALS; SCAVONE, CRISTOFORO; KAWAMOTO, ELISE MITIKO. Insulin and Autophagy in Neurodegeneration. FRONTIERS IN NEUROSCIENCE, v. 13, MAY 22 2019. Web of Science Citations: 0. (16/22996-9, 16/07427-8)
YSHII, LIDIA M.; MANFIOLLI, ADRIANA O.; DENADAI-SOUZA, ALEXANDRE; KINOSHITA, PAULA F.; GOMES, MARCELO D.; SCAVONE, CRISTOFORO. Tumor necrosis factor receptor-associated factor 6 interaction with alpha-synuclein enhances cell death through the Nuclear Factor-kB pathway. IBRO REPORTS, v. 9, p. 218-223, DEC 2020. Web of Science Citations: 0. (12/07784-4, 11/10303-5, 14/24951-7, 09/12375-3, 18/01308-2, 16/07427-8, 19/10044-1, 18/14289-6, 13/22196-4)
LEPSCH, LUCILIA B.; PLANETA, CLEOPATRA S.; SCAVONE, CRITOFORO. Cocaine Causes Apoptotic Death in Rat Mesencephalon and Striatum Primary Cultures. BIOMED RESEARCH INTERNATIONAL, 2015. Web of Science Citations: 10.
KAWAMOTO, E. M.; LIMA, L. S.; MUNHOZ, C. D.; YSHII, L. M.; KINOSHITA, P. F.; AMARAL, F. G.; PESTANA, R. R. F.; ORELLANA, A. M. M.; CIPOLLA-NETO, J.; BRITTO, L. R. G.; et al. Influence of N-methyl-D-aspartate receptors on ouabain activation of nuclear factor-kappa B in the rat hippocampus. Journal of Neuroscience Research, v. 90, n. 1, p. 213-228, JAN 2012. Web of Science Citations: 25.
DEGASPARI, SABRINA; TZANNO-MARTINS, CARMEN BRANCO; FUJIHARA, CLARICE KAZUE; ZATZ, ROBERTO; BRANCO-MARTINS, JOAO PAULO; VIEL, TANIA ARAUJO; BUCK, HUDSON DE SOUZA; MARQUES ORELLANA, ANA MARIA; BOEHMER, ANA ELISA; LIMA, LARISSA DE SA; et al. Altered KLOTHO and NF-kappa B-TNF-alpha Signaling Are Correlated with Nephrectomy-Induced Cognitive Impairment in Rats. PLoS One, v. 10, n. 5, MAY 11 2015. Web of Science Citations: 21.
VASCONCELOS, ANDREA RODRIGUES; KINOSHITA, PAULA FERNANDA; YSHII, LIDIA MITIKO; MARQUES ORELLANA, ANA MARIA; BOEHMER, ANA ELISA; LIMA, LARISSA DE SA; ALVES, ROSANA; ANDREOTTI, DIANA ZUKAS; MARCOURAKIS, TANIA; SCAVONE, CRISTOFORO; et al. Effects of intermittent fasting on age-related changes on Na,K-ATPase activity and oxidative status induced by lipopolysaccharide in rat hippocampus. NEUROBIOLOGY OF AGING, v. 36, n. 5, p. 1914-1923, MAY 2015. Web of Science Citations: 12. (11/10303-5)
YSHII, LIDIA M.; DENADAI-SOUZA, ALEXANDRE; VASCONCELOS, ANDREA R.; AVELLAR, MARIA CHRISTINA W.; SCAVONE, CRISTOFORO. Suppression of MAPK attenuates neuronal cell death induced by activated glia-conditioned medium in alpha-synuclein overexpressing SH-SY5Y cells. JOURNAL OF NEUROINFLAMMATION, v. 12, OCT 26 2015. Web of Science Citations: 5. (09/12375-3, 11/10303-5, 11/12255-8)
KAWAMOTO, E. M.; GLEICHMANN, M.; YSHII, L. M.; DE SA LIMA, L.; MATTSON, M. P.; SCAVONE, C.. Effect of activation of canonical Wnt signaling by the Wnt-3a protein on the susceptibility of PC12 cells to oxidative and apoptotic insults. Brazilian Journal of Medical and Biological Research, v. 45, n. 1, p. 58-67, JAN 2012. Web of Science Citations: 12. (06/59722-1)
KINOSHITA, PAULA F.; YSHII, LIDIA M.; ORELLANA, ANA MARIA M.; PAIXAO, AMANDA G.; VASCONCELOS, ANDREA R.; LIMA, LARISSA DE SA; KAWAMOTO, ELISA M.; SCAVONE, CRISTOFORO. Alpha 2 Na+,K+-ATPase silencing induces loss of inflammatory response and ouabain protection in glial cells. SCIENTIFIC REPORTS, v. 7, JUL 7 2017. Web of Science Citations: 2. (11/04327-9, 14/05026-0, 11/22844-0, 16/07376-4, 11/10303-5, 11/12255-8, 11/21308-8)
VASCONCELOS, ANDREA R.; YSHII, LIDIA M.; VIEL, TANIA A.; BUCK, HUDSON S.; MATTSON, MARK P.; SCAVONE, CRISTOFORO; KAWAMOTO, ELISA M.. Intermittent fasting attenuates lipopolysaccharide-induced neuroinflammation and memory impairment. JOURNAL OF NEUROINFLAMMATION, v. 11, MAY 6 2014. Web of Science Citations: 59. (06/57729-9, 09/11915-4)
MUNHOZ‚ C.D.; GLEZER‚ I.; KAWAMOTO‚ E.M.; ARAÚJO‚ A.P.N.; LEPSCHA‚ L.B.; PLANETA‚ C.S.; DELUCIA‚ R.; SCAVONE‚ C.. Changes in sodium‚ potassium-ATPase induced by repeated fencamfamine: the roles of cyclic AMP-dependent protein kinase and the nitric oxide-cyclic GMP pathway. Neuropharmacology, v. 45, n. 8, p. 1151-1159, 2003.
GLEZER‚ I.; MUNHOZ‚ C.D.; KAWAMOTO‚ E.M.; MARCOURAKIS‚ T.; AVELLAR‚ M.C.W.; SCAVONE‚ C.. MK-801 and 7-Ni attenuate the activation of brain NF-[kappa] B induced by LPS. Neuropharmacology, v. 45, n. 8, p. 1120-1129, 2003. (02/03506-8)
CABRAL-COSTA, J. V.; ANDREOTTI, D. Z.; MELLO, N. P.; SCAVONE, C.; CAMANDOLA, S.; KAWAMOTO, E. M.. Intermittent fasting uncovers and rescues cognitive phenotypes in PTEN neuronal haploinsufficient mice. SCIENTIFIC REPORTS, v. 8, JUN 5 2018. Web of Science Citations: 1. (14/18689-8, 13/20594-2, 16/22996-9, 15/25491-2, 11/21308-8)
MAZUCANTI, CAIO H.; KAWAMOTO, ELISA M.; MATTSON, MARK P.; SCAVONE, CRISTOFORO; CAMANDOLA, SIMONETTA. Activity-dependent neuronal Klotho enhances astrocytic aerobic glycolysis. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM, v. 39, n. 8, p. 1544-1556, AUG 2019. Web of Science Citations: 0. (16/07427-8, 16/09971-7)
CRUNFLI, FERNANDA; MAZUCANTI, CAIO HENRIQUE; MACEDO DE MORAES, RUAN CARLOS; COSTA, ANDRESSA PEREIRA; RODRIGUES, ALICE CRISTINA; SCAVONE, CRISTOFORO; TORRAO, ANDREA DA SILVA. NO-Dependent Akt Inactivation by S-Nitrosylation as a Possible Mechanism of STZ-Induced Neuronal Insulin Resistance. JOURNAL OF ALZHEIMER'S DISEASE, v. 65, n. 4, p. 1427-1443, 2018. Web of Science Citations: 2. (10/14090-3, 14/06372-0, 16/07427-8)
CARARO-LOPES, MARINA MINTO; YOKOYAMA MAZUCANTI, CAIO HENRIQUE; SCAVONE, CRISTOFORO; KAWAMOTO, ELISA MITIKO; BERWICK, DANIEL CHARLES. The relevance of alpha-KLOTHO to the central nervous system: Some key questions. AGEING RESEARCH REVIEWS, v. 36, p. 137-148, JUL 2017. Web of Science Citations: 13. (14/14199-6, 13/10787-8, 16/07427-8, 11/21308-8)
MUNHOZ, CAROLINA DEMARCHI; SORRELLS, SHAWN F.; CASO, JAVIER R.; SCAVONE, CRISTOFORO; SAPOLSKY, ROBERT M.. Glucocorticoids Exacerbate Lipopolysaccharide-Induced Signaling in the Frontal Cortex and Hippocampus in a Dose-Dependent Manner. JOURNAL OF NEUROSCIENCE, v. 30, n. 41, p. 13690-13698, OCT 13 2010. Web of Science Citations: 77.
KAWAMOTO, ELISA MITIKO; MUNHOZ, CAROLINA DEMARCHI; LEPSCH, LUCILIA BROCHADO; LIMA, LARISSA DE SA; GLEZER, ISAIAS; MARKUS, REGINA PEKELMANN; MARTINS DE SILVA, CLAUDIA LUCIA; CAMARINI, ROSANA; MARCOURAKIS, TANIA; SCAVONE, CRISTOFORO. Age-related changes in cerebellar phosphatase-1 reduce Na,K-ATPase activity. NEUROBIOLOGY OF AGING, v. 29, n. 11, p. 1712-1720, NOV 2008. Web of Science Citations: 7.
ARAUJO‚ A.P.N.; DELUCIA‚ R.; SCAVONE‚ C.; PLANETA‚ C.S.. Repeated predictable or unpredictable stress: effects on cocaine-induced locomotion and cyclic AMP-dependent protein kinase activity. Behavioural Brain Research, v. 139, n. 1, p. 75-81, 2003.
LEITE, J. A.; ISAKSEN, T. J.; HEUCK, A.; SCAVONE, C.; LYKKE-HARTMANN, K.. The alpha(2) Na+/K+-ATPase isoform mediates LPS-induced neuroinflammation. SCIENTIFIC REPORTS, v. 10, n. 1, AUG 25 2020. Web of Science Citations: 0. (16/07427-8, 16/21343-1, 14/10171-0)
KINOSHITA, PAULA FERNANDA; MARQUES ORELLANA, ANA MARIA; NAKAO, VINICIUS WATANABE; DE SOUZA PORT'S, NATACHA MEDEIROS; MENEZES QUINTAS, LUIS EDUARDO; KAWAMOTO, ELISA MITIKO; SCAVONE, CRISTOFORO. The Janus face of ouabain in Na+/K+-ATPase and calcium signalling in neurons. British Journal of Pharmacology, MAR 2021. Web of Science Citations: 0. (18/14289-6, 16/22996-9, 19/12974-6, 16/07427-8)
EYSERT, FANNY; KINOSHITA, PAULA FERNANDA; MARY, ARNAUD; VAILLANT-BEUCHOT, LOAN; CHECLER, FREDERIC; CHAMI, MOUNIA. Molecular Dysfunctions of Mitochondria-Associated Membranes (MAMs) in Alzheimer's Disease. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES, v. 21, n. 24, DEC 2020. Web of Science Citations: 1. (19/26084-2)
VAILLANT-BEUCHOT, LOAN; MARY, ARNAUD; PARDOSSI-PIQUARD, RAPHAELLE; BOURGEOIS, ALEXANDRE; LAURITZEN, INGER; EYSERT, FANNY; KINOSHITA, PAULA FERNANDA; CAZARETH, JULIE; BADOT, CELINE; FRAGAKI, KONSTANTINA; et al. Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimer's disease models and human brains. ACTA NEUROPATHOLOGICA, v. 141, n. 1, p. 39-65, JAN 2021. Web of Science Citations: 2. (18/14289-6)
DE MELLO, NATALIA PRUDENTE; ANDREOTTI, DIANA ZUKAS; ORELLANA, ANA MARIA; SCAVONE, CRISTOFORO; KAWAMOTO, ELISA MITIKO. Inverse sex-based expression profiles of PTEN and Klotho in mice. SCIENTIFIC REPORTS, v. 10, n. 1, NOV 19 2020. Web of Science Citations: 0. (11/21308-8, 16/07427-8, 16/22996-9, 15/25491-2)
(References retrieved automatically from State of São Paulo Research Institutions)
GLEZER, Isaias. Modulação da sinalização da resposta inflamatória no SNC pelo glutamato, óxido nítrico e receptor TLR4. 139 f. Tese (Doutorado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (00/10679-0)
MAZUCANTI, Caio Henrique Yokoyama. Efeitos da proteína Klotho sobre a sinalização de insulina e metabolismo energético no sistema nervoso central.. Tese (Doutorado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (13/10787-8)
VASCONCELOS, Andrea Rodrigues. Influência dos receptores TLR4 e TLR2 nos efeitos comportamentais e bioquímicos induzidos pela dieta intermitente em camundongos nocaute.. Tese (Doutorado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (11/12255-8)
ARAUJO, Ana Paula Natalini de. Aspectos Comportamentais e Moleculares da Sensibilização Cruzada entre Estresse e Cocaina.. Dissertação (Mestrado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (99/01620-3)
FRANCO, Lucas Augusto Moyses. Efeito da proteção desencadeada pelo estrógeno na linhagem C6 de glioma de rato. Dissertação (Mestrado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (08/56167-2)
YSHII, Lidia Mitiko. Efeitos da alfa-sinucleína na modulação da atividade do fator de transcrição nuclear <font face=\"Symbol\">kB em células SH-SY5Y.. Tese (Doutorado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (07/54291-5)
ORELLANA, Ana Maria Marques. Efeitos da administração intrahipocampal de ouabaína na modulação das vias NFκB, BDNF-CREB e WNT/β-catenina ao longo de um decurso temporal de 24 horas.. Tese (Doutorado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (11/22844-0)
LUIZ, Marcus Vinícius de Souza João. Comparação da efetividade e segurança de soluções de manutenção intermitente de cateter venoso central de longa permanência em pacientes submetidos à hemodiálise.. Tese (Doutorado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (13/07878-1)
CARARO, Marina Minto. Sinalização via receptor N-Metil-D-Aspartato e modulação da Na+, K+-ATPase em camundongos hipomórficos para klotho: efeitos em hipocampo e cerebelo.. Dissertação (Mestrado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (14/14199-6)
LEPSCH, Lucília Brochado. Toxicidade causada pela cocaína in vitro: participação da via dopaminérgica e do fator de transcrição NF-kB.. Tese (Doutorado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (02/13920-6)
VASCONCELOS, Andrea Rodrigues. Efeitos comportamentais e bioquímicos da dieta intermitente na vigência de um estímulo inflamatório no hipocampo de ratos.. Dissertação (Mestrado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (09/11915-4)
MALTA, Marília Brinati. Efeitos adaptativos induzidos pelo estresse crônico imprevisível nos receptores do fator liberador de corticotrofina tipo 2 e de glicocorticóides no sistema nervoso central de ratos.. Tese (Doutorado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (06/52566-4)
MAZUCANTI, Caio Henrique Yokoyama. Sinalização intracelular desencadeada por concentrações subtóxicas de estreptozotocina em células neuro-2A: modelo in vitro de neurodegeneração associada à doença de Alzheimer.. Dissertação (Mestrado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (10/14090-3)
KINOSHITA, Paula Fernanda. Sinalização inflamatória e a modulação da expressão de genes induzida pela ação da ouabaína nas isoformas a1, a2 - Na+, K+- ATPase em células da glia.. Dissertação (Mestrado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (11/04327-9)