CV Lattes
ResearcherID
Universidade de São Paulo (USP). Faculdade de Medicina de Ribeirão Preto (FMRP) (Institutional affiliation for the last research proposal) Birthplace: Brazil
bachelor's at Odontologia from Faculdade de Odontologia de Araçatuba Unesp (2003), master's at Fisiologia from Faculdade de Medicina de Ribeirão Preto - USP (2008) and doctorate at Fisiologia from Faculdade de Medicina de Ribeirão Preto - USP (2013). Has experience in Physiology, focusing on Physiology, acting on the following subjects: estresse, reparação óssea, tíbia de rato, diazepam and xerostomia. (Source: Lattes Curriculum)
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Nowadays, chronic pain is one of the most important health problems. Trials are hardly handled trying to elucidate tissue, cellular and molecular events responsible for triggering this illness. This could make a difference for efficient treatments needed. Succinate is a product of GABA metabolization, a compound of Krebs' circle and a GPR91 ligand. GPR91 was considered as an orphan recept…
Neuropathic pain is an intractable, chronic syndrome that may arise from injury to peripheral nerves and is associated with allodynia, hyperalgesia, spontaneous pain, repetitive discharge of nociceptors and the expansion of receptive fields of nociceptive input. Our understanding of the pathophysiology of this debilitating syndrome is very limited and represents the main obstacle to the d…
Chronic pain treatment is still a challenge to researchers and clinicians. Much has been elucidated regarding to the physiopathology of several painful conditions, but less has been developed aiming new drugs to mainly control neuropathic pain. Also, much has explored about phenotypic changes caused by genetic and protein expression shift in nociceptive system, but nothing has been mentio…
| 4 / 3 | Completed scholarships in Brazil |
| 1 / 1 | Completed scholarships abroad |
| 5 / 4 | All Scholarships |
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KUSUDA, RICARDO; CARREIRA, ELEONORA UCHOA; ULLOA, LUIS; CUNHA, FERNANDO QUEIROZ; KANASHIRO, ALEXANDRE; CUNHA, THIAGO MATTAR. Choline attenuates inflammatory hyperalgesia activating nitric oxide/cGMP/ATP-sensitive potassium channels pathway. Brain Research, v. 1727, JAN 15 2020. (12/23846-0, 13/08216-2, 11/20343-4, 10/16823-8)