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Thais Girão da Silva

CV Lattes

Secretaria da Saúde (São Paulo - Estado). Hospital das Clínicas da Faculdade de Medicina da USP (HCFMUSP). Instituto do Coração Professor Euryclides de Jesus Zerbini (INCOR)  (Institutional affiliation for the last research proposal)
Birthplace: Brazil

Graduated in Biomedical Sciences on Federal University of Sao Paulo (2005). In 2009 did Master in Molecular Biology at the same University. PhD at University of Sao Paulo (2018). Has experience in Cardiovascular Biology research, acting on the following topics: cardiovascular cell differentiation, endothelial to mesenchymal transition, vein graft, endothelial cell, stem cells, cytoskeleton remodeling and myocardial infarction. Currently working with development of new therapies approaches for cardiac regeneration. (Source: Lattes Curriculum)

Scholarships in Brazil
FAPESP support in numbers * Updated November 16, 2019
Total / Available in English
3 / 1 Completed scholarships in Brazil

Associated processes
Most frequent collaborators in research granted by FAPESP
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Use this Research Supported by FAPESP (BV/FAPESP) channel only to send messages referring to FAPESP-funded scientific projects.





Keywords used by the researcher
Scientific publications resulting from Research Grants and Scholarships under the grantee's responsibility (2)

(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)

Data from Web of Science

GIRAO-SILVA, THAIS; BASSANEZE, VINICIUS; GASTALHO CAMPOS, LUCIENE CRISTINA; BARAUNA, VALERIO GARRONE; OLIVEIRA DALLAN, LUIS ALBERTO; KRIEGER, JOSE EDUARDO; MIYAKAWA, AYUMI AUREA. Short-term mechanical stretch fails to differentiate human adipose-derived stem cells into cardiovascular cell phenotypes. BIOMEDICAL ENGINEERING ONLINE, v. 13, . Web of Science Citations: 9. (07/58942-0)

MIYAKAWA, AYUMI A.; GIRAO-SILVA, THAIS; KRIEGER, JOSE E.; EDELMAN, ELAZER R.. Rapamycin activates TGF receptor independently of its ligand: implications for endothelial dysfunction. Clinical Science, v. 132, n. 4, p. 437-447, . Web of Science Citations: 2. (14/06844-9, 13/17368-0)

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