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Camila Oliveira de Souza

CV Lattes

Universidade de São Paulo (USP). Instituto de Ciências Biomédicas (ICB)  (Institutional affiliation for the last research proposal)
Birthplace: Brazil

Over the course of my research career I have been extremely fascinated about the link between alterations on inflammatory state and metabolism. During my master?s (State University of Londrina, Brazil) I was focused in understand the protective role of non-steroids anti-inflammatory drugs (NSAIDs) on liver abnormalities triggered by insulin resistance associated to cancer cachexia. Later on my PhD (University of Sao Paulo, Brazil), I dedicated myself to unravel the properties of the palmitoleic acid (POA), in cellular biology and function of macrophages and hepatocytes from obese mice. This omega-7 fatty acid modulated macrophages polarization and protected the liver from insulin resistance and chronic inflammation associated with obesity. In a further mechanistic characterization, I observed the liver metabolic function were dependent of PPAR activation, but POA anti-inflammatory role occurred in whole liver or macrophages independently of PPAR nuclear receptors, PPAR and PPAR . Since 2018 I have started my postdoctoral training with Dr. Dayoung Oh (UT Southwestern, Dallas, TX) as my mentor and we are exploring the role of GPR92 on the type 2 diabetes induced by diet. GPR92 is a G-Protein Coupled Receptor highly expressed in immune cells and pancreas, which seems to have an important metabolic/anti-inflammatory role that needs to be further explored especially due to the drugability potential of this GPCR. (Source: Lattes Curriculum)

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Scientific publications resulting from Research Grants and Scholarships under the grantee's responsibility (7)

(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)

Data from Web of Science

SILVEIRA, LOREANA S.; PIMENTEL, GUSTAVO D.; SOUZA, CAMILA O.; BIONDO, LUANA A.; TEIXEIRA, ALEXANDRE ABILIO S.; LIMA, EDSON A.; BATATINHA, HELENA A. P.; ROSA NETO, JOSE C.; LIRA, FABIO S.. Effect of an acute moderate-exercise session on metabolic and inflammatory profile of PPAR- knockout mice. Cell Biochemistry and Function, v. 35, n. 8, p. 510-517, . Web of Science Citations: 4. (13/25310-2, 14/01246-6, 13/10861-3, 13/04765-1, 13/09367-4)

DE SOUZA, CAMILA O.; VANNICE, GRETCHEN K.; ROSA NETO, JOSE C.; CALDER, PHILIP C.. Is Palmitoleic Acid a Plausible Nonpharmacological Strategy to Prevent or Control Chronic Metabolic and Inflammatory Disorders?. MOLECULAR NUTRITION & FOOD RESEARCH, v. 62, n. 1, . Web of Science Citations: 17. (13/04765-1, 16/01409-8)

TEIXEIRA, ALEXANDRE ABILIO DE S.; LIRA, FABIO S.; PIMENTEL, GUSTAVO D.; DE SOUZA, CAMILA OLIVEIRA; BATATINHA, HELENA; BIONDO, LUANA A.; YAMASHITA, ALEX S.; LIMA JUNIOR, EDSON A.; ROSA NETO, JOSE C.. Aerobic Exercise Modulates the Free Fatty Acids and Inflammatory Response During Obesity and Cancer Cachexia. CRITICAL REVIEWS IN EUKARYOTIC GENE EXPRESSION, v. 26, n. 3, p. 187-198, . Web of Science Citations: 8. (13/25310-2, 13/09367-4)

SOUZA, CAMILA O.; TEIXEIRA, ALEXANDRE A. S.; BIONDO, LUANA A.; SILVEIRA, LOREANA S.; CALDER, PHILIP C.; ROSA NETO, JOSE C.. Palmitoleic acid reduces the inflammation in LPS-stimulated macrophages by inhibition of NF kappa B, independently of PPARs. Clinical and Experimental Pharmacology and Physiology, v. 44, n. 5, p. 566-575, . Web of Science Citations: 14. (13/04765-1, 16/01409-8)

TEIXEIRA, ALEXANDRE ABILIO S.; BIONDO, LUANA AMORIM; SILVEIRA, LOREANA S.; LIMA, EDSON A.; BATATINHA, HELENA A.; DINIZ, TIEGO A.; DE SOUZA, CAMILA OLIVEIRA; COMIN, JEFERSON; ROSA NETO, JOSE CESAR. Doxorubicin modulated clock genes and cytokines in macrophages extracted from tumor-bearing mice. CANCER BIOLOGY & THERAPY, . Web of Science Citations: 0. (15/16777-0, 19/09854-9, 16/01409-8)

DE SOUZA, CAMILA O.; TEIXEIRA, ALEXANDRE A. S.; BIONDO, LUANA A.; LIMA JUNIOR, EDSON A.; BATATINHA, HELENA A. P.; ROSA NETO, JOSE C.. Palmitoleic Acid Improves Metabolic Functions in Fatty Liver by PPAR-Dependent AMPK Activation. Journal of Cellular Physiology, v. 232, n. 8, p. 2168-2177, . Web of Science Citations: 16. (13/04765-1, 16/01409-8)

DE SOUZA, CAMILA OLIVEIRA; VALENZUELA, CARINA A.; BAKER, ELLA J.; MILES, ELIZABETH A.; NETO, JOSE C. ROSA; CALDER, PHILIP C.. Palmitoleic Acid has Stronger Anti-Inflammatory Potential in Human Endothelial Cells Compared to Oleic and Palmitic Acids. MOLECULAR NUTRITION & FOOD RESEARCH, v. 62, n. 20, . Web of Science Citations: 2. (13/04765-1, 16/01409-8, 15/06376-8)

SOUZA, CAMILA O.; TEIXEIRA, ALEXANDRE A. S.; BIONDO, LUANA AMORIM; SILVEIRA, LOREANA SANCHES; DE SOUZA BREDA, CRISTIANE N.; BRAGA, TARCIO T.; CAMARA, NIELS O. S.; BELCHIOR, THIAGO; FESTUCCIA, WILLIAM T.; DINIZ, TIEGO A.; et al. Palmitoleic acid reduces high fat diet-induced liver inflammation by promoting PPAR-gamma-independent M2a polarization of myeloid cells. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR AND CELL BIOLOGY OF LIPIDS, v. 1865, n. 10, . Web of Science Citations: 0. (13/04765-1, 19/06172-4, 19/24112-9, 15/19530-5, 16/01409-8)

Academic Publications

(References retrieved automatically from State of São Paulo Research Institutions)

SOUZA, Camila Oliveira de. Papel dos PPARs sobre os benefícios imuno-metabólicos promovidos pelo ácido palmitoleico.. Tese (Doutorado) -  Instituto de Ciências Biomédicas.  Universidade de São Paulo (USP).  São Paulo.  (13/04765-1

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