CV Lattes
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Universidade de São Paulo (USP). Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto (FFCLRP) (Institutional affiliation for the last research proposal) Birthplace: Brazil
Professor of Graduation Program in Neurology and Neuroscience in the Faculty of Medicine of Ribeirão Preto (FMRP) since 2014 and Senior Professor at Psychology department of Faculty of Philosophy Sciences and Letters of Ribeirão Preto (FFCLRP) since 2020 and University of São Paulo (USP). Graduated in Biological Sciences from Universidade de São Paulo (USP), Master at Pharmacology from Universidade de São Paulo and PhD at Physiology from Universidade de São Paulo. Post Graduation in Wake Forest University, USA (1990-1991) and Universitaet Tuebingen, Alemanha (1992-1995). She was Professor (1985-1990) in the Universidade Federal de Santa Catarina and in the Faculdade de Odontologia de Ribeirão Preto da USP (1999-2020). She was member of undergraduation SBFIS commission (2010-2019) and of Teaching Comission of APS (2015-2018) and recently was nominated (2018) member of the Board General Assembly of International Union of Physiological Sciences (IUPS) where is co-chair of the Education Commitee since 2015. She was also Secretary of Neuroscience Institute in Ribeirão Preto from 2016 -2021 when started to work with scientific divulgation. As a scientific researcher got several CNPq and FAPESP grants and gave several national and international lectures including Germany, France, Chile, USA, Germany, Japan, China and New Zealand.She organized national and international events in physiology teaching and neuroscience and published text and articles in education. As researcher has experience in Neurosciences in the Pharmacology, Physiology and Experimental Odontology working more recently with neuroimmunoendocrine and behaviour alterations during inflammatory process and neurodegenerative. Guest Editor of the Journal of Neuroendocrinology, hold several awards, one in the CNPq /Renama, Academic Production category. For contact mjrocha@forp.usp.brf (Source: Lattes Curriculum)
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Sepsis, a systemic inflammation triggered by an infectious agent, produces an inflammatory state in the central nervous system (CNS) that leads to autonomic, endocrine and cognitive-behavioral dysfunctions observed in clinical and experimental studies. Previous findings from this laboratory suggest that oxidative stress, microglial activation, synaptic dysfunction and eventually apoptosis…
Sepsis and its consequences, septic shock and organ dysfunction, pose a serious public health problem. Besides the high costs, cause great mortality and the survivors after hospital discharge may have cognitive impairments resulting in future admissions burdening even more the national health system. During sepsis are released cytokines, prostaglandins, leukotrienes, nitric oxide, etc., w…
(Only some records are available in English at this moment)
The installation of an inflammatory process in the CNS, even in the absence of in situ infection, is commonly referred to as sepsis-associated encephalopathy (SAE). This condition is responsible for brain dysfunction that will result in functional and cognitive impairment in sepsis surviving patients. Although the cognitive disabilities found in sepsis survivors should not be equated with…
Peripheral inflammatory mediators that reach the Central Nervous System (CNS) trigger the activation of microglial cells resulting in neuroinflammation. This inflammatory response may cause changes in the permeability of the blood-brain barrier (BBB), which with other alterations may lead to brain dysfunction causing the appearance of long-term neurological sequelae. Studies have shown th…
(Only some records are available in English at this moment)
Worldwide, more than 30 million people are affected by sepsis, which is a life-threatening organ dysfunction caused by a dysregulated host response to infection. Several studies suggest that sepsis might act as a significant inflammatory "hit" that potentially increases the susceptibility of the brain to neurodegenerative disease and the development of dementia later in life. Survivors of…
Sepsis, a systemic inflammatory condition triggered by an infectious agent, induces an inflammatory status in central nervous system (CSN) which leads to autonomic, cognitive and behavioral alterations. The paraventricular nucleus of the hypothalamus (PVN) plays a major role in endocrine control during sepsis and is associated to the regulation of neurohypophyseal hormones and HPA axis. O…
Sepsis is a potentially fatal condition that occurs when the body produces an inflammatory response to an infectious agent (bacteria, fungi, parasites or viruses) that can lead to multiple organ failure, shock and death. In the pathophysiological process of sepsis excessive production and release of inflammatory mediators may directly or indirectly activate the central nervous system. In …
| 12 / 9 | Completed research grants |
| 45 / 16 | Completed scholarships in Brazil |
| 1 / 1 | Ongoing scholarships abroad |
| 3 / 2 | Completed scholarships abroad |
| 61 / 28 | All research grants and scholarships |
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