Abstract
The infection of the gastric mucosa by Helicobacter pylori (H. pylori) promotes a chronic inflammation, providing a microenvironment favorable for the appearance of atrophic gastritis with progression to gastric cancer. The increase in IL-6 synthesis, as well as the presence of the polymorphisms -174 (G/C) (rs1800795), -572 (C/G) (rs1800796) and -597 (G/A) (rs1800797), appear to be associ…