- Research Grants
He is bachelor?s at Physical Education in the School of Physical Education and Sport of Catanduva (2001), postgraduates ?lato sensu" in Sport Medicine in the School of Medicine of Catanduva (2003) and Masters (2009) and Ph.D. (2013) in Physiology in Ribeirão Preto Medical School (FMRP), USP. In 2015, he was visitant researcher in Dr. Ronald Kahn's lab (Joslin Diabetes Center - Harvard Medical School, USA). Between 2011 and 2016, Between 2011 and 2013, he performed research internship (doctorate and post-doc) in the laboratory of Dr. Marco Sandri at the Venetian Institute of Molecular Medicine (VIMM, Padua, Italy) and University of Padua. Between 2013 and 2019, he developed post-doctoral activities in Dept. of Biochemistry and Physiology (FMRP) using a multidisciplinary approach, such as histology, biochemistry and cellular and molecular biology, to elucidate: 1) the in vivo contribution of IGF-1/insulin signaling for the effects of beta2-adrenergic agonists on protein metabolism, tropism, function and plasticity of rodent skeletal muscles and 2) the relationship between atrophy signalings and muscular adaptative effects of physical training. Currently, he is Adjunct Professor in the Department of Physical Exercise at School of Physical Education, Physiotherapy and Occupational Therapy (EEFFTO) - Federal University of Minas Gerais (UFMG). He has experience in the area of ​​General Physiology, Biochemistry and Cellular and Molecular Biology, with an emphasis on endocrinology and metabolism, acting on the following topics: skeletal muscle proteolytic systems, protein synthesis, beta2-adrenergic agonists, intracellular signalling, physical exercise, models of muscle atrophy and hypertrophy and in vivo and in vitro gene manipulation. (Source: Lattes Curriculum)
The knowledge of therapeutic strategies able to counteract skeletal muscle atrophy is an important issue for human health. In previous studies, we have showed that b2-adrenoceptors (AR) activation regulates muscle protein metabolism through the suppression of the activity of ubiquitin (Ub)-proteasome system, the major proteolytic system involved in muscle wasting, and the expression of ...
The development of therapeutic strategies capable of inhibiting protein breakdown and preserving muscle mass is an important issue for human health. In previous studies, we have showed that beta2-adrenoceptors (AR) activation mitigates muscle atrophy through the suppression of the activity of ubiquitin (Ub)-proteasome (UPS) and autophagy-lysosomal (ALS) systems, the major proteolytic sy...
(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)
|Data from Web of Science|