Abstract
Chronic consumption of ethanol (EtOH) and abstinence to it has been associated to disorders such as anxiety and depression. Several brain structures and neurotransmitter systems seem to be involved in mediating this effect. In animal models non-specific periferic or intracerebral (Dorsal Raphe Nucleus) blockade of Nitric Oxide Synthetase (NOS) attenuates the anxiogenic effects induced by …