CV Lattes
ResearcherID
Universidade de São Paulo (USP). Faculdade de Ciências Farmacêuticas de Ribeirão Preto (FCFRP) (Institutional affiliation from the last research proposal) Birthplace: Brazil
Graduated in Pharmacy at Universidade do Extremo Sul Catarinense (2009), Master's (2011) and PhD (2015) in Sciences with emphasis in Pharmacology by the Department of Pharmacology of the Medical School of Ribeirão Preto, University of São Paulo (FMRP / USP). Post-doctorate in neuropsychopharmacology at the Translational Neuropsychiatry Unit, Department of Clinical Medicine, University of Aarhus (Aarhus, Denmark - 05/2015 to 10/2017). Reviewer of six scientific journals and associate editor of the journal Acta Neuropsychiatrica (Cambridge University Press). Experience in the field of Pharmacy, with emphasis on Pharmacology, working mainly on the following topics: neuropsychopharmacology, neurobiology of depression and mechanism of action of fast acting antidepressants. Acted as a Medical Science Liaison in different pharmaceutical industries (Johnson Johnson, Biogen, and Servier) with a focus on neuroscience/psychiatry/neurology and oncology. Experience in translating clinical research documents such as the investigator's brochure, protocol, informed consent form, among others. (Source: Lattes Curriculum)
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Glutamate, through activation of NMDA receptors, is capable of enhancing the nitric oxide (NO) synthesis. Over the last years, the involvement of the NMDA-NO pathway in the neurobiology of depression has been described. The use of NMDA antagonists, like ketamine, or NO synthesis inhibitors, such as 7-NI, produce antidepressant-like effects in animals, reinforcing the potential of glutamat…
Several data suggest the participation of glutamatergic and nitrergic systems in the neurobiology of depression. The administration of glutamatergic antagonists (ketamine) or nitric oxide (NO) inhibitors (7-NI) in animals induces antidepressant-like effects. The effects of ketamine seem to be dependent of neural plasticity mechanisms, because the injection of protein synthesis inhibitor …
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