Abstract
The higher pathogenicity of falciparum malaria is ascribed to cytoadhesion of the P. falciparum-infected erythrocytes (Pf-iEs) in the microvasculature of vital organs (e.g. brain, lungs and placenta). Although this phenomenon is not yet noted for P. vivax, clinical complications similar to those observed for severe falciparum malaria are now described to P. vivax, suggesting that key path…