Abstract
During obesity, there is an increase in the renin-angiotensin system (RAS) activity. Thus, a greater RAS activation, and the consequent increased levels of peripheral angiotensin II (ANG II), can activate forebrain areas of the central nervous system (CNS) free of the blood brain barrier located in the lamina terminalis, and rich in ANG II receptors, such as the subfornical organ (SFO). W…