CV Lattes
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Universidade de São Paulo (USP). Faculdade de Medicina de Ribeirão Preto (FMRP) (Institutional affiliation from the last research proposal) Birthplace: Brazil
Bachelor degree in Biomedicine at Londrina State University, working on innate immunity recognition of fungal infection, using plant-derived lectins to stimulate innate and adaptive immune response. Master?s degree at Dr João Santana Silva?s laboratory working in the field of immunity against protozoan parasites, Leishmania. PhD in Basic and Applied Immunology at Ribeirão Preto Medical School, University of São Paulo at Dr Dario Simões Zamboni's lab, working with inflammasome activation and its role in driving resistance or susceptibility to Leishmania infection. Also evaluating the mechanisms of pathogen control and host response during lung infection. Visiting Scholar as PhD Student in University of California, Berkeley, under supervision of Prof Russell E Vance. (Source: Lattes Curriculum)
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Visceral leishmaniasis (VL) is a disease caused by Leishmania infantum. The sandfly vector inoculates parasites in the skin, which disseminate to organs such as lymph nodes, spleen, liver and bone marrow, hindering the function of these organs and causing symptomatic disease. VL can be fatal if not treated. However, a large proportion of endemic individuals do not develop any disease. The…
Leishmania amazonensis is the causative agent of American Tegumentary Leishmaniasis in Brasil. The infected individuals can develop several types of the disease, such as benign forms presenting localized lesions that spontaneously heal (Localized cutaneous Leishmaniasis - LCL), the diffuse form of disease, characterized by high parasite numbers in non-ulcerated lesions (Diffuse cutaneous …
The genus Leishmania is composed by several protozoans which cause a complex of diseases called leishmaniasis. Visceral leishmaniasis (VL) is a systemic, chronic and potentially fatal disease caused by L. infantum, endemic in Brazil. This parasite is transmitted through the bite of phlebotomies insects, and promastigotes inoculated rapidly infect macrophages, disseminating to the spleen, …
The innate immune system is composed of several sensors and effectors widely expressed in macrophages, dendritic cells and epithelial cells. These molecules can rapidly induce inflammatory responses by triggering the production of cytokines, chemokines and release of danger signals present in the cytoplasm. In this context, host cell death has been reemerged as an important effector mecha…
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