CV Lattes
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Universidade Estadual de Campinas (UNICAMP). Instituto de Biologia (IB) (Institutional affiliation from the last research proposal) Birthplace: Brazil
I hold a Bachelor's and a Licentiate degree in Biological Sciences from the University of Campinas (UNICAMP, 2018), where I also completed my Master's in Functional and Molecular Biology, with an emphasis on Physiology (2021). During my undergraduate studies, I undertook an internship at the Centre for Biomedical Research (CBMR) at the University of Algarve, Portugal (2016-2017). Currently, I am pursuing a Ph.D. in the Molecular and Morphofunctional Biology program at UNICAMP, working at the Obesity and Comorbidities Research Center (OCRC). I also completed a research internship at the Joslin Diabetes Center (Harvard Medical School) in Boston (2023-2024). My research focuses on Physiology, particularly Endocrine Physiology, with an emphasis on obesity, adipose tissue metabolism, thermogenesis, Alzheimer's disease, and hypothalamic dysregulation. (Source: Lattes Curriculum)
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Obesity is defined as an excessive and abnormal accumulation of fat and is often associated with hypothalamic dysregulation. The consumption of high-fat diets has been considered the main factor responsible for inducing hypothalamic inflammation, commonly found in obese individuals. This inflammation has been associated with chronic hypothalamic resistance to the signs of anorexigenic and…
Alzheimer's is a neurodegenerative disease caused by the accumulation and extracellular deposition of the amyloid beta protein in the hippocampus. This process results in neuronal death, leading to memory impairment and progressive cognitive decline. Aging is the main risk factor for the development of Alzheimer's, and the hippocampus is severely affected in this condition. Recent studies…
ARHGAP21 controls multiple cellular functions, such as migration, proliferation, differentiation and vesicles intracellular traffic. ARHGAP21 is a molecular partner of ARFs GTPases, controlling the formation of vesicles from the Golgi. Our ARHGAP21-heterozygous animal model (Het, 50% of the protein expression) treated with the high fat diet (HFD) had higher content of triglycerides in the…
Activation of brown adipose tissue (BAT) increases energy expenditure (EE), and its activity is inversely correlated with body mass index and fat mass. BAT generates heat in response to cold exposure due to its unique expression of uncoupling protein 1 (UCP1) that dissipates energy by uncoupling the proton motive force from ATP production. Although UCP1 expression is restricted to BAT und…
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