Abstract
Type 1 Diabetes mellitus (DM1) is a pathology that occurs due to autoimmune destruction of pancreatic ß cells, initially without clinical manifestations. This autoimmune response is mediated by different proinflammatory cytokines secreted by an islet immune cell infiltrate, such as interleukin (IL) 1ß, interferon (IFN) and tumor necrosis factor (TNF), in process known as insulitis. In res…