CV Lattes
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Universidade Estadual de Campinas (UNICAMP). Instituto de Biologia (IB) (Institutional affiliation from the last research proposal) Birthplace: Brazil
Biotechnologist, Master in Bioprocesses (UFSCar) and PhD in Sciences (Immunology) from USP. Works in the area of #8203;#8203;immunometabolism with a focus on investigating the causes of mitochondrial dysfunction in macrophages. Recently, he revealed the role of the mitoKATP ion channel in the development of tolerance to LPS by macrophages. Currently, he studies the mechanisms mediated by HIF-1a responsible for mitochondrial dysfunction in macrophages activated by LPS. (Source: Lattes Curriculum)
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Mitochondrial dynamics are essential for cellular adaptation, yet their regulation in immune cells and impact on immune responses remain poorly understood. This study investigates the role of hypoxia-inducible factor-1a (HIF-1a) in the development of mitochondrial dysfunction during macrophage-mediated inflammation. Preliminary findings indicate that HIF-1a stabilization occurs prior to m…
The Obesity epidemic already affects more than 650 million people worldwide and predisposes to the development of several comorbidities, such as Atherosclerosis and Type 2 Diabetes. During Obesity, macrophages infiltrate adipose tissue that assume a pro-inflammatory and contribute to low-grade local and systemic inflammation and subsequent insulin resistance. Blood hypercholesterolemia le…
Activation of the NLRP3 inflammasome follows a "two-step" route: priming and activation. It has been a consensus that the activation step involves a greater efflux of K+, however the mechanism by which the efflux of K+ induces the activation of the inflammasome is not clear yet. Recent studies have shown the essential role of mitochondrial stress in leaking ox-mtDNA into the cytoplasm to …
| 1 / 1 | Ongoing scholarships in Brazil |
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