Bachelor's Degree in Biomedical Sciences from the Institute of Biomedical Sciences (ICB) at the University of São Paulo (USP), currently a PhD student in the Immunology Graduate Program at ICB-USP. During my academic training, I dedicated four years to scientific initiation, focusing on research in immunometabolism, particularly mitophagy and inflammatory processes. I was awarded a FAPESP Scientific Initiation Fellowship (Process 2022/13122-6), which allowed me to deepen my knowledge and contribute to advancing this area of research. Additionally, I participated in an International Research Internship at the Laboratory of Tissue Biology at Yale University, supported by FAPESP (Process 2023/07057-0). During this experience, I expanded my understanding of immune mechanisms, with a focus on skin aging. Currently, as a PhD student, I am part of the Transplant Immunobiology Laboratory at ICB-USP. My doctoral research investigates the effects of interleukin 11 (IL-11) on the inflammaging process and its metabolic impact on keratinocytes. (Source: Lattes Curriculum)
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The skin plays essential roles for the body, including acting as a protective barrier and providing immune surveillance. Its aging is marked by structural and functional changes that lead to metabolic and phenotypic alterations. An important aspect of this process is cellular senescence, where cells secrete factors from the senescence-associated secretory phenotype (SASP), including pro-i…
Psoriasis is an autoimmune disease characterized by hyperproliferation of keratinocytes which also promote chronic inflammation in the skin. Furthermore, the inflammatory process is triggered by coordinated action of infiltrating immune cells in the skin and keratinocytes, where the latter release exacerbated levels of antimicrobial peptides, cytokines and chemokines. It is suggested that…
Psoriasis is a chronic autoimmune disease characterized by inflammation of the skin resulting from hyperproliferation of keratinocytes. IL-17A is a cytokine that plays a critical role in the inflammatory loop mediated by keratinocytes. We aim to investigate whether IL-17A induces mitochondrial damage in keratinocytes and how mitochondrial quality control mechanisms, such as mitophagy, cou…
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