Abstract
Thirst and sodium appetite are regulated by central facilitatory mechanisms (activated by angiotensin II, mineralocorticoid or hyperosmolarity) and by central inhibitory mechanisms such as oxytocinergic and lateral parabrachial nucleus (LPBN) mechanisms. Injections of the GABAA agonist muscimol into the LPBN of normohydrated rats induce an intense sodium intake that is reduced by the bloc…