Abstract
Our group demonstrated that both S100A9 and a synthetic peptide identical to the C-terminal portion of murine S100A9 (mS100A9p) induced an antinociceptive effect in inflammatory pain models. In addition, mS100A9p inhibited the functions of mouse adherent peritoneal cells, which are critical for interaction with both malignant and stromal cells in tumor microenvironment. Tumor growth does …