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BCR-ABL-mediated regulation of SH3BGRL2, D53, PRAME, DAP12 and calcineurin A beta and biological consequences of this regulation on CML

Grant number: 05/58764-0
Support type:Scholarships in Brazil - Doctorate (Direct)
Effective date (Start): April 01, 2006
Effective date (End): November 30, 2009
Field of knowledge:Biological Sciences - Immunology - Cellular Immunology
Principal researcher:João Gustavo Pessini Amarante Mendes
Grantee:Daniel Diniz de Carvalho
Home Institution: Instituto de Ciências Biomédicas (ICB). Universidade de São Paulo (USP). São Paulo , SP, Brazil
News published in Agência FAPESP Newsletter about the scholarship:
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Scientific publications
(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)
DE CARVALHO, D. D.; BINATO, R.; PEREIRA, W. O.; LEROY, J. M. G.; COLASSANTI, M. D.; PROTO-SIQUEIRA, R.; BUENO-DA-SILVA, A. E. B.; ZAGO, M. A.; ZANICHELLI, M. A.; ABDELHAY, E.; CASTRO, F. A.; JACYSYN, J. F.; AMARANTE-MENDES, G. P. BCR-ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients. Oncogene, v. 30, n. 2, p. 223-233, Jan. 2011. Web of Science Citations: 30.
Academic Publications
(References retrieved automatically from State of São Paulo Research Institutions)
. BCR-ABL-mediated regulation of SH3BGRL2, D53, PRAME, DAP12 e Calcineurin A beta and biological consequences of this regulation on CML.. 2009. Doctoral Thesis - Universidade de São Paulo (USP). Instituto de Ciências Biomédicas (ICB/SDI) São Paulo.

Please report errors in scientific publications list by writing to: cdi@fapesp.br.