BCR-ABL-mediated regulation of SH3BGRL2, D53, PRAME, DAP12 and calcineurin A beta and biological consequences of this regulation on CML

• Grant number: 05/58764-0
• Support type: Scholarships in Brazil - Doctorate (Direct)
• Effective date (Start): April 01, 2006
• Effective date (End): November 30, 2009
• Field of knowledge: Biological Sciences - Immunology - Cellular Immunology
• Principal researcher: João Gustavo Pessini Amarante Mendes
• Grantee: Daniel Diniz de Carvalho
• Home Institution: Instituto de Ciências Biomédicas (ICB). Universidade de São Paulo (USP). São Paulo , SP, Brazil

Scientific publications

(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)

DE CARVALHO, D. D.; BINATO, R.; PEREIRA, W. O.; LEROY, J. M. G.; COLASSANTI, M. D.; PROTO-SIQUEIRA, R.; BUENO-DA-SILVA, A. E. B.; ZAGO, M. A.; ZANICHELLI, M. A.; ABDELHAY, E.; CASTRO, F. A.; JACYSYN, J. F.; AMARANTE-MENDES, G. P. BCR-ABL-mediated upregulation of PRAME is responsible for knocking down TRAIL in CML patients. Oncogene, v. 30, n. 2, p. 223-233, Jan. 2011. Web of Science Citations: 30.

Academic Publications

(References retrieved automatically from State of São Paulo Research Institutions)

. BCR-ABL-mediated regulation of SH3BGRL2, D53, PRAME, DAP12 e Calcineurin A beta and biological consequences of this regulation on CML.. 2009. Doctoral Thesis - Universidade de São Paulo (USP). Instituto de Ciências Biomédicas (ICB/SDI) São Paulo.