Characterization of endogenous NOD2 agonist isolated from synovial fluids of patients with rheumatoid arthritis.

• Grant number: 10/19770-2
• Support Opportunities: Scholarships in Brazil - Post-Doctoral
• Start date: March 01, 2011
• End date: December 31, 2014
• Field of knowledge: Biological Sciences - Immunology
• Principal Investigator: Fernando de Queiroz Cunha
• Grantee: Rafael Freitas de Oliveira Franca
• Host Institution: Faculdade de Medicina de Ribeirão Preto (FMRP). Universidade de São Paulo (USP). Ribeirão Preto , SP, Brazil
• Associated scholarship(s): 12/14924-7 - Role of NOD2 signaling and molecular mechanisms involved in the generation of IL-17 pathogenic T cells in autoimmune diseases, BE.EP.PD

Scientific publications

(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)

FRANCA, R. F. O.; VIEIRA, S. M.; TALBOT, J.; PERES, R. S.; PINTO, L. G.; ZAMBONI, D. S.; LOUZADA-JUNIOR, P.; CUNHA, F. Q.; CUNHA, T. M.. Expression and activity of NOD1 and NOD2/RIPK2 signalling in mononuclear cells from patients with rheumatoid arthritis. SCANDINAVIAN JOURNAL OF RHEUMATOLOGY, v. 45, n. 1, p. 8-12, JAN 2 2016. (10/19770-2, 13/08216-2, 11/19670-0)

FRANCA, RAFAEL F. O.; COSTA, RENATA S.; SILVA, JAQUELINE R.; PERES, RAPHAEL S.; MENDONCA, LEILA R.; COLON, DAVID F.; ALVES-FILHO, JOSE CARLOS; CUNHA, FERNANDO Q.. IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system. JOURNAL OF NEUROINFLAMMATION, v. 13, JUN 22 2016. (10/19770-2, 13/08216-2, 11/19670-0)