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Priapism and voiding dysfunction in sickle cell disease: pathophysiology and new drug candidates

Grant number: 18/06243-6
Support type:Scholarships in Brazil - Young Researchers
Effective date (Start): April 01, 2018
Effective date (End): July 31, 2019
Field of knowledge:Biological Sciences - Pharmacology - Autonomic Pharmacology
Principal researcher:Fábio Henrique da Silva
Grantee:Fábio Henrique da Silva
Home Institution: Centro de Hematologia e Hemoterapia (HEMOCENTRO). Universidade Estadual de Campinas (UNICAMP). Campinas , SP, Brazil
Associated research grant:17/08122-9 - Priapism and voiding dysfunction in Sickle Cell Disease: pathophysiology and new drug candidates, AP.JP
News published in Agência FAPESP Newsletter about the scholarship:
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Scientific publications
(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)
MUSICKI, BILJANA; ANELE, UZOMA A.; CAMPBELL, JEFFREY D.; KARAKUS, SERKAN; SHIVA, SRUTI; SILVA, FABIO H.; BURNETT, ARTHUR L. Dysregulated NO/PDE5 signaling in the sickle cell mouse lower urinary tract: Reversal by oral nitrate therapy. Life Sciences, v. 238, DEC 1 2019. Web of Science Citations: 0.
KARAKUS, SERKAN; ANELE, UZOMA A.; SILVA, FABIO H.; MUSICKI, BILJANA; BURNETT, ARTHUR L. Urinary dysfunction in transgenic sickle cell mice: model of idiopathic overactive bladder syndrome. AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY, v. 317, n. 3, p. E540-E546, SEP 2019. Web of Science Citations: 1.
SILVA, FABIO HENRIQUE; FERTRIN, KLEBER YOTSUMOTO; ALEXANDRE, EDUARDO COSTA; CALMASINI, FABIANO BERALDI; FRANCO-PENTEADO, CARLA FERNANDA; COSTA, FERNANDO FERREIRA. Impairment of Nitric Oxide Pathway by Intravascular Hemolysis Plays a Major Role in Mice Esophageal Hypercontractility: Reversion by Soluble Guanylyl Cyclase Stimulator. Journal of Pharmacology and Experimental Therapeutics, v. 367, n. 2, p. 194-202, NOV 1 2018. Web of Science Citations: 1.

Please report errors in scientific publications list by writing to: cdi@fapesp.br.