CV Lattes
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Universidade Federal de São Paulo (UNIFESP). Campus São Paulo. Escola Paulista de Medicina (EPM) (Institutional affiliation from the last research proposal) Birthplace: Brazil
Dr. Foresto-Neto holds a degree in Biological Sciences from the Mackenzie Presbyterian University, scientific initiation in Renal Pathophysiology from the Faculty of Medicine at the University of São Paulo (Supervisor: Dr. Clarice Kazue Fujihara), and a PhD in Nephrology from the Faculty of Medicine at the University of São Paulo (Supervisor: Prof. Dr. Roberto Zatz), with an internship period in Munich, Germany, at the Ludwig-Maximilians-Universität München (Supervisor: Prof. Dr. Hans-Joachim Anders). He completed his postdoctoral research at the Institute of Biomedical Sciences at the University of São Paulo (Supervisor: Prof. Dr. Niels Olsen Saraiva Câmara), and at Mayo Clinic, in Rochester, MN, USA (Mentor: Prof. Dr. Michael F. Romero). Received a Level 5 Technical Training Fellowship (FAPESP) at the Department of Immunology, Institute of Biomedical Sciences, University of São Paulo, for the implementation of a confocal microscopy system. Currently serves as a FAPESP Young Investigator at the Division of Nephrology, Department of Medicine, Federal University of São Paulo; as a Collaborating Researcher at Mayo Clinic (Rochester, MN, USA); and as an Early Career Editor Fellow for the American Journal of Physiology-Renal Physiology. (Source: Lattes Curriculum)
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The insect Malpighian tubule (MT) is composed of principal cells [the most abundant cell type that transport ions from hemolymph to the tubule lumen] and stellate cells [responsible for Cl- and water movement]. The MT is functionally homologous to the mammalian kidneys and several similarities allow Drosophila to serve as a dynamic in vivo model to study translational scenarios. The Droso…
Renal disease and the consequent need for renal replacement therapy represent a serious global public health problem. The mechanisms involved in the progression of the chronic kidney disease (CKD) remain incompletely understood, which makes it difficult to develop effective strategies for its treatment. Recent studies suggest that the progression of CKD and the development of fibrosis are…
Diabetic nephropathy is one of the most important causes of end-stage renal disease around the world, as a result of 1) the growing prevalence of Type 2 diabetes mellitus, which in turn reflects the increasing prevalence of overweight and obesity, as well as aging of the world's population; 2) increased survival of diabetic patients thanks to numerous therapeutic advances. The mechanisms …
(Only some records are available in English at this moment)
Sudden increase in serum oxalate levels can result in renal calcium oxalate (CaOx) crystals formation, which is a well-known cause of kidney injury. It has been reported that NLRP3 inflammasome activation, necroptosis via RIPK3-MLKL complex, and mitochondrial damage are involved in CaOx-related tubular injury. Changes in renal cell metabolism may also participate in the pathogenesis of Ca…
Diabetic nephropathy (DN) is one of the most important causes of end-stage renal disease around the world. The mechanisms that lead to the development of DN are complex, and despite progress in recent decades, are still far from being fully understood. Inflammation is involved in both the pathogenesis and progression of DN. There is evidence that activation of innate immunity by diabetes …
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| 8 / 8 | All research grants and scholarships |
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