CV Lattes
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Universidade de São Paulo (USP). Faculdade de Medicina (FM) (Institutional affiliation from the last research proposal) Birthplace: Brazil
Graduated in Veterinary Medicine at the Federal University of Bahia (UFBA), Master in Experimental Pathology at UFBA and PhD in Sciences by the Experimental and Comparative Pathology Program at the Faculty of Veterinary Medicine and Animal Science at the University of São Paulo (USP). He was Substitute Professor of the subjects Embriology and Histology of Domestic Animals and Human Histology of the Department of Biomorphology at the Federal University of Bahia and Assistant Professor of the disciplines of Animal Body, Biological Processes, Aggression and Defense at the University of Salvador (UNIFACS / Laureate). He has scientific and teaching experience in the areas of Immunology, Immunoparasitology, Cell Biology, Molecular Biology, Immunogenetics, Genomics, Molecular Epidemiology, Histopathology and Biomorphology. (Source: Lattes Curriculum)
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Exploration of the genetic basis of Visceral Leishmaniasis (VL) has identified substantial amounts of biomarkers and thus has contributed to improve understanding of the pathogenesis of the VL. However, such findings have not been converted into disease control strategies. The identification of immunotherapeutic targets depends on the identification of metabolic pathways of the host, cons…
Visceral leishmaniasis (VL) is the most severe form of leishmaniasis. The disease affects mainly the cells of the fagocitic mononuclear system in bone marrow, spleen, liver, lungs, intestine and skin and can be fatal if not be treated. The incidence of about 90% of cases of VL is concentrated in five countries, including Brazil. Dogs are considered the main domestic reservoir of Leishmani…
Genetic diversity of the host-parasite interplay may be associated with differences in disease outcome, resistance to treatment and eco-epidemiological traits. Deletion of a >12kb loci on chromosome 31 of L. infantum has been associated with resistance to treatment in visceral leishmaniasis (VL). Named Mitelfosine Sensitive Loci (MSL), this chromosomal segment includes genes related to vi…
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