Dagmar Ruth Stach - Machado
CV Lattes
Universidade Estadual de Campinas (UNICAMP). Instituto de Biologia (IB) (Institutional affiliation for the last research proposal) Birthplace: Brazil
Dagmar Ruth Stach - Machado in research grants and scholarships supported by FAPESP.
- Sequential IL-23 and IL-17 and Increased Mmp8 and Mmp14 expression characterize the progression of an experimental model of periodontal disease in type 1 diabetes, PUB.ART
Abstract
Molecular mechanisms responsible for periodontal disease (PD) and its worsening in type 1 Diabetes Mellitus (DM1) remain unknown. Cytokine profile and expression levels of collagenases,Mmp14, and tissue inhibitors were determined, as were the numbers of neutrophils and macrophages in combined streptozotocin-induced DM1 and ligature-induced PD models. Increased IL-23 (80-fold) and Mmp8ex...
- Characterization of the inflammatory and immune mechanisms involved in periodontal disease in rats obese, AP.R
Abstract
Periodontal disease is characterized as a group of lesions in the tissues that surround and support teeth in their sockets, which can result in tooth loss. The main etiologic agent of periodontal disease is bacterial plaque, but the host immune response determines disease susceptibility. Although the host response means to be protective, it initiates a cascade of events that can lead to...
- Changes in MMPs and inflammatory cells in experimental gingivitis, PUB.ART
Abstract
In periodontal disease, extensive disorganization of the extracellular matrix promotes the loss of adhesion between the teeth and periodontium. A previous study suggested a reduction in the area occupied by collagen in the gingiva, during the first week of periodontal disease induction, however, the remaining fibers were more compact and thicker. Therefore, it was decided to investigate...
- Evaluation of cell migration and chemokine expression in the progression of periodontal disease in obese and normal mice, BP.IC
Abstract
Periodontal disease is characterized a group of lesions in the tissues that surround and support teeth in their sockets, which can result in tooth loss. The main etiologic agent of periodontal disease is bacterial plaque, but the host response that determines disease susceptibility. Although the host response has meant to be protective, it initiates a cascade of events that can lead to ...
- Analysis of mechanisms involved in periodontal tissue destruction on obese and obese diabetic rats with experimental periodontal disease, BP.MS
- Avaliacion of Type I Diabetes mellitus in migracion profile of inflammatory cells and cytokines during the induced periodontal dissease progression, BP.IC
(Only some records are available in English at this moment)
| 8 / 3 | Completed research grants |
| 13 / 4 | Completed scholarships in Brazil |
| 21 / 7 | All research grants and scholarships |
| Associated processes | |
(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)
| Publications | 4 |
| Citations | 71 |
| Cit./Article | 17.8 |
| Data from Web of Science | |
SILVA, JULIETE A. F.; FERRUCCI, DANILO L.; PERONI, LUIS A.; ABRAHAO, PATRICIA G. S.; SALAMENE, ALINE F.; ROSSA-JUNIOR, CARLOS; CARVALHO, HERNANDES F.; STACH-MACHADO, DAGMAR R.. Sequential IL-23 and IL-17 and increased Mmp8 and Mmp14 expression characterize the progression of an experimental model of periodontal disease in type 1 diabetes. Journal of Cellular Physiology, v. 227, n. 6, p. 2441-2450, JUN 2012. Web of Science Citations: 22.
OBANDO PEREDA, G.; CORNEJO FUDINAGA, A. C.; SALAS BELTRAN, H.; PERONI, L. A.; STACH-MACHADO, D.. Inflammatory and bone regulators expression in murine macrophages under exposure of commercial and experimental mineral trioxide aggregate. AUSTRALIAN DENTAL JOURNAL, v. 57, n. 3, p. 284-291, SEP 2012. Web of Science Citations: 4.
OBANDO-PEREDA, G. A.; FISCHER, L.; STACH-MACHADO, D. R.. Titanium and zirconia particle-induced pro-inflammatory gene expression in cultured macrophages and osteolysis, inflammatory hyperalgesia and edema in vivo. Life Sciences, v. 97, n. 2, p. 96-106, MAR 3 2014. Web of Science Citations: 27.
SILVA, JULIETE APARECIDA F.; FERRUCCI, DANILO LOPES; PERONI, LUIS ANTONIO; ISHI, EDUARDO DE PAULA; ROSSA-JUNIOR, CARLOS; CARVALHO, HERNANDES F.; STACH-MACHADO, DAGMAR RUTH. Periodontal Disease-Associated Compensatory Expression of Osteoprotegerin Is Lost in Type 1 Diabetes Mellitus and Correlates with Alveolar Bone Destruction by Regulating Osteoclastogenesis. Cells Tissues Organs, v. 196, n. 2, p. 137-150, 2012. Web of Science Citations: 18.
(References retrieved automatically from State of São Paulo Research Institutions)
PEREDA, Gustavo Alberto Obando. Envolvimento dos TLR2 e TLR4 no reconhecimento das partículas de titânio e zircônia por macrofagos murinos. 2012. Tese (Doutorado) – Instituto de Biologia. Universidade Estadual de Campinas (UNICAMP).
BIANCHI, Henrique de Oliveira. Análise dos mecanismos envolvidos na destruição tecidual periodontal em ratos obesos com doença periodontal experimental. 2012. Dissertação (Mestrado) - Instituto de Biologia. Universidade Estadual de Campinas (UNICAMP).
DIAS, Leticia Chaves Ferreira. Caracterização imunoquímica dos anticorpos monoclonais que reconhecem proteínas do capsídeo viral do vírus da tristeza dos citrus do complexo Capão Bonito. 2002. xix, 94f. Dissertação (Mestrado) - Instituto de Biologia. Universidade Estadual de Campinas (UNICAMP). Campinas.