Abstract
The Phosphatydilinositol 3-kinase (PI3K/Akt) pathway is altered in many human cancers and it is widely studied in many glandular tumors as, breast and prostate carcinomas. In relation to salivary gland neoplasia, previous studies have been demonstrated dysregulation of PI3K signaling as, Akt overexpression, in adenoid cystic carcinoma. Glycogen Synthase Kinase 3 (GSK3) is a well-establish…