Luiz Felipe Souza e Silva, also known as Silva L.F.S, is a highly qualified and skilled professional. He holds a broad academic background, having completed his Bachelor's degree in Pharmacy at Faculdades Metropolitanas Unidas (FMU) in 2013. Later, he obtained his Master's degree in Health Sciences from Faculdade de Ciências Médicas da Santa Casa de São Paulo, with a project carried out between 2016 and 2018. More recently, he completed his PhD in Pharmacology from the University of São Paulo, Institute of Biomedical Sciences (USP/ICB), with a project carried out between 2019 and 2022. Throughout his academic career, Luiz Felipe has acquired expertise in the analysis of mitochondria and mental disorders. Furthermore, he has extensive experience in scientific research, having participated in several research projects and published numerous articles in internationally renowned scientific journals. With his academic background and research experience, Luiz Felipe Souza e Silva is a highly qualified and well-prepared professional to work in various areas related to Pharmacy and Health Sciences. Dr. Luiz Felipe Souza e Silva is presently involved in a postdoctoral research project at the esteemed Laboratory of Clinical Immunology and Allergy (LIM60) located in the University of São Paulo - School of Medicine (INCOR - HCFMUSP). Here, he is conducting an extensive study on evaluating the mitochondrial function of cardiomyocytes derived from induced pluripotent stem cells (iPSCMs) of patients diagnosed with chronic Chagas cardiomyopathy. (Source: Lattes Curriculum)
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About 30% of Chagas disease patients develop chronic Chagas disease cardiomyopathy (CCC), an inflammatory cardiomyopathy that occurs decades after the initial infection, while most infected patients (60%) remain asymptomatic in the so-called indeterminate form (IF). Myocardial fibrosis, inflammation and mitochondrial dysfunction are involved in the arrhythmia substrate and triggering even…
Among all hypotheses related to Schizophrenia (SZ), we can highlight the neurodevelopment theory. According to it, environmental factors, such as hypoxia, could cause alterations and abnormalities in cellular machinery, including modifications in neural proliferation, migration and differentiation, in addition to changes in synapsis formation, which could lead to degenerative processes. H…
The goal of this project is to investigate the role of sirtuins (SIRTs), specifically sirtuin 1, nuclear, and 3, mitochondrial, in mitochondrial dysfunction due to hypoxia in different animal and cellular models of Schizophrenia (SHZ): astrocytes exposed to CoCl2 and from spontaneous hypertensive rats, SHR. The significant value of this project is the fact that it is known that one of the…
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