Abstract
Ninety percent of the Congenital Adrenal Hyperplasia (CAH) cases present the 21-hydroxylase deficiency resulting in an impaired Cortisol secretion and, in some cases an impaired aldosterone secretion. Then, an increased production of steroid precursors occurs secondary to ACTH accumulation by missing negative feedback in the Pituitary gland. In the Adrenal, these precursors are deviated …