CV Lattes
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Universidade de São Paulo (USP). Instituto de Ciências Biomédicas (ICB) (Institutional affiliation from the last research proposal) Birthplace: Brazil
Biomedical scientist with an MSc in Genetics (UFPE) and a PhD in Clinical Oncology, Stem Cells, and Cell Therapy (FMRP/USP). Completed a postdoctoral fellowship at the Hematology Branch of the NHLBI/NIH (Bethesda, MD, USA; 20212023) and served as a research associate at UFPE (20232025). Currently a postdoctoral researcher at ICB/USP, investigating molecular vulnerability mechanisms as therapeutic targets in acute myeloid leukemia. His research focuses on the molecular pathophysiology of hematological malignancies and clonal conditions, with emphasis on the identification of diagnostic, prognostic, and therapeutic markers in leukemias, bone marrow failure syndromes, and hemoglobinopathies. (Source: Lattes Curriculum)
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Acute myeloid leukemia (AML) is characterized by the clonal expansion of immature myeloid cells, resulting in hematopoietic failure, with symptoms such as anemia and infections. Clinical outcomes are unfavorable, with an estimated overall survival inferior to 25% in five years. AML cells exhibit metabolic reprogramming that promotes dependence on oxidative phosphorylation for ATP producti…
The JAK2V617F mutation is identified in the majority of patients with myeloproliferative neoplasms (MPN), and is considered a diagnostic hallmark for these diseases. Additional mutations in MPL and CALR, which participates of JAK/STAT pathways, are diagnostic markers for 80% of JAK2 negatives patients. Inhibitors of JAK / STAT pathway represent a therapeutic advance in MPN treatment, howe…
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