Abstract
Chronic thromboembolic pulmonary hypertension (TEPH) is characterized by the formation of intraluminal thrombus leading to right ventricular overload. It is characterized by the presence of anticardiolipin antibodies, factor VIII, macrophage chemo-attracting protein, endothelin-1 and low levels of thromobomodulin [1]. The first line of treatment is anticoagulation and endarterectomy (PEA)…