Abstract
Alzheimer's disease (AD) is a neurodegenerative disease triggered by several factors, including the accumulation of ²-amyloid peptides (pA²), which leads to loss of synapses and neurons. These peptides are generated from the proteolytic cleavage of the amyloid precursor protein (APP) and can modulate a neuronal activity. Subsequently, these peptides can be degraded in the brain or drained…