Abstract
The molecular etiopathogenesis of adrenocortical tumors (CT) is partially known. Some genes and pathways are already consolidated and their functions are well established, such as TP53, via WNT / BETA-CATENIN, YAP1 and via SHH. However, the total clarification of the mechanisms involved in adrenal tumorigenesis is still lacking. In this context, the tumor suppression gene SAMD9, target of…