Breno Picin Casagrande

CV Lattes ResearcherID ORCID Google Scholar Citations

Universidade Federal de São Paulo (UNIFESP). Campus Baixada Santista. Instituto de Saúde e Sociedade (ISS) (Institutional affiliation from the last research proposal)
Birthplace: Brazil

Ph.D. student in Sciences (FAPESP Fellow) in the Interdisciplinary Health Sciences Graduate Program (PPGICS) (CAPES: 5) at the Federal University of São Paulo, Baixada Santista Campus (UNIFESP-BS), with a research exchange period (BEPE - FAPESP) at the University of Southampton, Southampton, United Kingdom. Holds a Master's degree in Sciences (CAPES Fellow) from the PPGICS (CAPES: 5) at UNIFESP-BS. Earned a Bachelor's degree in Nutrition from UNIFESP-BS, including a study exchange period (CAPES) through the Science Without Borders Program in partnership with Athlone Institute of Technology (AIT), Ireland. Member of the Endocrine Metabolic Physiology Laboratory (LaFEM) at UNIFESP-BS. (Source: Lattes Curriculum)

News published in Agência FAPESP Newsletter about the researcher

More items Less items

TITULO

Articles published in other media outlets ( ):

More items Less items

VEICULO: TITULO (DATA)

Scholarships in Brazil

Ongoing (most recent)

Abstract
Low-quality obesogenic diets are estimated to account for more than one-fifth of the world's deaths. This inadequate dietary pattern and increased adiposity are related to several metabolic alterations and complications, and most of them have an inflammatory background. Also, some behavioural changes are associated with its consumption. It is necessary to introduce a balanced diet in term…
Strategy to minimize the adverse effects of replacing an obese diet with a standard diet on behavioral and neuroendocrine parameters in an animal model, BP.DR

Scholarships abroad

Completed (most recent)

Abstract
Obesogenic diets (ODs) induce adiposity increase and obesity-like features, such as inflammation, in humans and animal models. OD causes inflammation increase and barrier dysfunction in the small intestine. Toll-like receptor 4 (TLR4) inflammatory cascade is the main pathway associated with these changes. OD leads to dysbiosis which is followed by the increase in lipopolysaccharides (LPS)…
Ascorbic acid as a strategy to improve inflammation and gut barrier disruption in a Caco-2 model: the role of butyric acid, BE.EP.DR

Virtual Library in numbers * Updated data on July 19, 2025

Total / Available in English

1 / 1	Ongoing scholarships in Brazil
1 / 1	Completed scholarships abroad
2 / 2	All Scholarships

Associated processes

Most frequent collaborators in research granted by FAPESP

Debora Estadella

Keywords used by the researcher

Biological Sciences Interdisciplinary Subjects Physiology

Please report errors on the page using this form.

Short URL

Abstract

Abstract