CV Lattes
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Secretaria da Saúde (São Paulo - Estado). Hospital das Clínicas da Faculdade de Medicina da USP (HCFMUSP) (Institutional affiliation from the last research proposal) Birthplace: Brazil
Medicine graduation by the Medical School of the University of Minas Gerais (2007-2013). Medical Residency in Internal Medicine at Instituto Mario Penna (2014-2016). Medical Residency in Endocrinology and Metabology at Santa Casa de Misericórdia in Belo Horizonte (2016-2018). Specialist degree in Endocrinology and Metabology by the Brazilian Society of Endocrinology and Metabology obtained in 2018. Volunteer in the Pediatric Endocrinology Sector of Childrens Institute - Hospital das Clínicas of Medical School - University of São Paulo (July to November, 2018). Doctorate degree in Endocrinology started in June 2019 at University of São Paulo (CNPq Scholarship # 140289/2020-8 January/2020 to November/2021; FAPESP Scholarship # 2021/12205-2 since December 2021). Research Fellowship (FAPESP Grant #2022/04870-9) at the Department of Endocrinology and Metabology of Brigham and Womens Hospital - Harvard Medical School, Boston, US (July/2022 to February/2023). (Source: Lattes Curriculum)
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The association between central precocious puberty (CPP) and polycystic ovary syndrome (PCOS) is suggested by epidemiological, pathophysiological, and genetic data; but there is still a lack of studies that robustly corroborate this association. Both disorders are linked to unfavorable metabolic and reproductive outcomes in adulthood, but it is not known whether the association of the two…
Precocious Puberty is defined as the appearance of secondary sex characteristics before 8 years of age in girls and before 9 years of age in boys, and leads to rapid advancement of skeletal maturation, causing loss of potential adult height, besides emotional and psychosocial impact. The most common mechanism of precocious puberty is the early activation of pulsatile GnRH secretion, known…
Central precocious puberty (CPP) is caused by premature re-emergence of pulsatile hypothalamic gonadotropin-releasing hormone (GnRH) secretion. The recognition of the genetic influence in this disturb has significantly increased after the description of loss-of-function mutations in two maternally imprinted genes (MKRN3 and DLK1) causing paternally transmitted CPP. The mechanisms of react…
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