The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH analogue therapy not restored by dihydrotestosterone administration

Jorge‚ A.A.L.; Souza‚ S.C.A.L.; Arnhold‚ I.J.P.; Mendonca‚ B.B.

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(Reference retrieved automatically from Google Scholar through information on FAPESP grant and its corresponding number as mentioned in the publication by the authors.)

The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH analogue therapy not restored by dihydrotestosterone administration

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Author(s):	Jorge‚ A.A.L. ; Souza‚ S.C.A.L. ; Arnhold‚ I.J.P. ; Mendonca‚ B.B. Total Authors: 4
Document type:	Journal article
Source:	Clinical Endocrinology; v. 60, n. 1, p. 36-40, 2004.

FAPESP's process:	00/14092-4 - Molecular diagnosis of the alterations in the GHRH-GH-IGF-1 axis in patients with short stature
Grantee:	Ivo Jorge Prado Arnhold
Support Opportunities:	Research Projects - Thematic Grants

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