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Investigation of the mechanisms involved in the susceptibility to Trypanosoma cruzi infection in phosphatidylinositol 3-kinase gamma (PI3Kgama) deficient mouse

Grant number: 11/09488-0
Support Opportunities:Scholarships in Brazil - Doctorate
Effective date (Start): September 01, 2011
Effective date (End): September 30, 2015
Field of knowledge:Biological Sciences - Immunology - Cellular Immunology
Principal Investigator:Thiago Mattar Cunha
Grantee:Maria Cláudia da Silva
Host Institution: Faculdade de Medicina de Ribeirão Preto (FMRP). Universidade de São Paulo (USP). Ribeirão Preto , SP, Brazil
Associated scholarship(s):13/24312-1 - The role of AKT1 and Akt2 signaling during Trypanosoma cruzi infection, BE.EP.DR
News published in Agência FAPESP Newsletter about the scholarship:
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Scientific publications
(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)
SILVA, MARIA C.; DAVOLI-FERREIRA, MARCELA; MEDINA, TIAGO S.; SESTI-COSTA, RENATA; SILVA, GRACE K.; LOPES, CARLA D.; CARDOZO, LUCAS E.; GAVA, FABIO N.; LYRONI, KONSTANTINA; DIAS, FABRICIO C.; et al. Canonical PI3K gamma signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis. NATURE COMMUNICATIONS, v. 9, . (13/08216-2, 12/14524-9, 11/09488-0)

Please report errors in scientific publications list by writing to: cdi@fapesp.br.