Investigation of the mechanisms involved in the susceptibility to Trypanosoma cruzi infection in phosphatidylinositol 3-kinase gamma (PI3Kgama) deficient mouse

• Grant number: 11/09488-0
• Support Opportunities: Scholarships in Brazil - Doctorate
• Effective date (Start): September 01, 2011
• Effective date (End): September 30, 2015
• Field of knowledge: Biological Sciences - Immunology - Cellular Immunology
• Principal Investigator: Thiago Mattar Cunha
• Grantee: Maria Cláudia da Silva
• Host Institution: Faculdade de Medicina de Ribeirão Preto (FMRP). Universidade de São Paulo (USP). Ribeirão Preto , SP, Brazil
• Associated scholarship(s): 13/24312-1 - The role of AKT1 and Akt2 signaling during Trypanosoma cruzi infection, BE.EP.DR

Scientific publications

(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)

SILVA, MARIA C.; DAVOLI-FERREIRA, MARCELA; MEDINA, TIAGO S.; SESTI-COSTA, RENATA; SILVA, GRACE K.; LOPES, CARLA D.; CARDOZO, LUCAS E.; GAVA, FABIO N.; LYRONI, KONSTANTINA; DIAS, FABRICIO C.; et al. Canonical PI3K gamma signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis. NATURE COMMUNICATIONS, v. 9, APR 17 2018. (13/08216-2, 12/14524-9, 11/09488-0)