CV Lattes
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Universidade de São Paulo (USP). Instituto de Ciências Biomédicas (ICB) (Institutional affiliation for the last research proposal) Birthplace: Brazil
Associate Professor - Institute of Biomedical Sciences and School of Medicine at University of Sao Paulo, Brazil. Associate Professor - Stanford University School of Medicine CA-USA. Founder and director - SPARK-Brasil. Research interest - cell metabolism. Member - Brazilian Academy of Sciences, São Paulo Academy of Sciences and Global Young Academy. (Source: Lattes Curriculum)
Mitochondria are key organelles involved in ATP production and regulation of several signaling cascades that ultimately affect cell death and survival. Mitochondrial dysfunction plays a critical role in the development and progression of cardiovascular diseases, such as myocardial infarction. Therefore, mitochondria have become an attractive therapeutic target for the treatment of cardiov…
4-hydroxy-2-nonenal (4-HNE) is a major by-product of lipid peroxidation, a process that is exacerbated under oxidative stress conditions. This aldehyde is highly reactive with proteins, lipids and DNA, causing target loss of function and degradation in most cases. For this reason, accumulation of 4-HNE has been correlated with the establishment and progression of many diseases, including …
Our current results demonstrate that intracellular protein quality control [machinery that detects, repairs and disposes cytotoxic proteins] is impaired during muscle dysfunction and wasting. We also show that pharmacological activation of β2-adrenoceptor induces an overall improvement of protein quality control-related mechanisms in skeletal muscle. Our next step is to establish a causal…
The cellular protein quality control (PQC) machinery detects repairs and disposes cytotoxic proteins through different proteolytic systems (i.e. ubiquitin-proteasome system and autophagy). We recently demonstrated that improving PQC protects against cardiac diseases. Here, we are going to characterize the PQC profile as well as the benefits of sustained ²2-adrenoceptor activation, a posit…
(Only some records are available in English at this moment)
Mitochondria are organelles that continually undergo fusion and fission. These opposite processes work together to maintain the shape, size, number and function of mitochondria. Recently, our group demonstrated that reestablishing mitochondrial fusion-fission balance through exercise or drug therapy is sufficient to restore mitochondrial bioenergetics and improves the prognosis of heart f…
Heart failure is a degenerative syndrome that affects over 20 million people worldwide, being considered an important health public problem. Overall, 50% of heart failure patients die in the time-window of 5 years after diagnostics. Heart failure is classified into 2 classes: heart failure with reduced ejection fraction (HFrEF) and heart failure with preserved ejection fraction (HFpEF). H…
Aldehydes are highly reactive molecules in biological systems, where they bind to biomolecules such as DNA and proteins, resulting in functional jeopardy. Aldehydic attack may have an exogenous source, when directly absorbed from the environment, or be generated endogenously, after ethanol metabolism (generating acetaldehyde) or during mitochondrial lipid peroxidation (forming 4-hydroxyno…
(Only some records are available in English at this moment)
Currently affecting 8 million people, mainly in Latin America (endemic area), Chagas Disease (CD) is caused by the protozoan Tryoanosoma cruzi (T. cruzi). The 2 medicines (Benznidazole and Nifurtimox) avaliable to treat CD focus on killing the parasite and are partially effective only in the acute phase of the disease. Based on this, new targets and therapies are needed. It was recently d…
Sustained loss of skeletal muscle mass and function occurs mainly during degenerative processes. Increased proteolytic activity (i.e. autophagy) has been extensively associated with muscle atrophy. However, proteolysis is also crucial for the maintenance of cellular homeostasis. In fact, autophagy serves as a protective system against non-functional proteins/organelles through a sophistic…
Despite scientific and therapeutic advances, ischemic heart disease is still the leading cause of death worldwide. This pathology is caused by decreased blood flow (ischemia), which promotes tissue damage and cell death according to the intensity and time of ischemia, thus causing biochemical and functional changes to the myocardium. Among these changes, the increase in intracellular calc…
(Only some records are available in English at this moment)
Geroscience has been successful in identifying evolutionarily conserved genes and molecular mechanisms capable of increasing longevity, mainly in the nematode Caenorhabditis elegans (C. elegans). However, it is still not clear whether this prolonged period of life is associated with improved healthspan (either relative or absolute). Healthspan is defined by the period of life in which an …
Doxorubicin is an anthracycline widely used to treat different types of cancer including leukemia, lymphomas and breast cancer. However, it has high risk of cardiotoxicity since it accumulates and negatively affects cardiac mitochondrial proteostasis and bioenergetics. Disruption of mitochondrial proteostasis triggers a conserved signaling pathway termed mitochondrial unfolded protein res…
The health benefits of exercise have long been recognized. The importance of autophagy for this process is now also well-established. New research suggests that significant benefits from exercise depend on selective degradation of defective mitochondria through mitophagy, but the mechanisms connecting these processes are unknown. This project aims to fill this critical gap in our knowledg…
(Only some records are available in English at this moment)
| 1 / 1 | Ongoing research grants |
| 5 / 5 | Completed research grants |
| 4 / 4 | Ongoing scholarships in Brazil |
| 20 / 20 | Completed scholarships in Brazil |
| 8 / 8 | Completed scholarships abroad |
| 38 / 38 | All research grants and scholarships |
| Associated processes | |
(References retrieved automatically from Web of Science and SciELO through information on FAPESP grants and their corresponding numbers as mentioned in the publications by the authors)
| Publications | 42 |
| Citations | 987 |
| Cit./Article | 23.5 |
| Data from Web of Science | |
FERREIRA, JULIO C. B.; MOREIRA, JOSE B. N.; CAMPOS, JULIANE C.; PEREIRA, MARCELO G.; MATTOS, KATT C.; COELHO, MARCELE A.; BRUM, PATRICIA C.. Angiotensin receptor blockade improves the net balance of cardiac Ca2+ handling-related proteins in sympathetic hyperactivity-induced heart failure. Life Sciences, v. 88, n. 13-14, p. 578-585, MAR 28 2011. Web of Science Citations: 17. (09/03143-1, 05/59740-7)
FERREIRA, JULIO C. B.; MOCHLY-ROSEN, DARIA. Nitroglycerin Use in Myocardial Infarction Patients - Risks and Benefits. CIRCULATION JOURNAL, v. 76, n. 1, p. 15-21, JAN 2012. Web of Science Citations: 32. (09/03143-1)
DISATNIK, MARIE-HELENE; HWANG, SUNHEE; FERREIRA, JULIO C. B.; MOCHLY-ROSEN, DARIA. New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases. JOURNAL OF MOLECULAR MEDICINE-JMM, v. 93, n. 3, p. 279-287, MAR 2015. Web of Science Citations: 9. (12/05765-2)
SUN, LIHAN; CESAR, JULIO; FERREIRA, BATISTA; MOCHLY-ROSEN, DARIA. ALDH2 Activator Inhibits Increased Myocardial Infarction Injury by Nitroglycerin Tolerance. Science Translational Medicine, v. 3, n. 107, NOV 2 2011. Web of Science Citations: 43. (09/03143-1)
PALANIYANDI, SURESH SELVARAJ; BATISTA FERREIRA, JULIO CESAR; BRUM, PATRICIA CHAKUR; MOCHLY-ROSEN, DARIA. PKC beta II inhibition attenuates myocardial infarction induced heart failure and is associated with a reduction of fibrosis and pro-inflammatory responses. JOURNAL OF CELLULAR AND MOLECULAR MEDICINE, v. 15, n. 8, p. 1769-1777, AUG 2011. Web of Science Citations: 17. (06/56321-6)
PAULINO, ELLENA CHRISTINA; BATISTA FERREIRA, JULIO CESAR; BECHARA, LUIZ ROBERTO; TSUTSUI, JEANE MIKE; MATHIAS, JR., WILSON; LIMA, FABIO BESSA; CASARINI, DULCE ELENA; CICOGNA, ANTONIO CARLOS; BRUM, PATRICIA CHAKUR; NEGRAO, CARLOS EDUARDO. Exercise Training and Caloric Restriction Prevent Reduction in Cardiac Ca2+-Handling Protein Profile in Obese Rats. Hypertension, v. 56, n. 4, p. 629-U137, OCT 2010. Web of Science Citations: 34. (09/03143-1)
FERREIRA, JULIO C. B.; BACURAU, ALINE V.; BUENO JUNIOR, CARLOS R.; CUNHA, TELMA C.; TANAKA, LEONARDO Y.; JARDIM, MAIRA A.; RAMIRES, PAULO R.; BRUM, PATRICIA C.. Aerobic exercise training improves Ca2+ handling and redox status of skeletal muscle in mice. Experimental Biology and Medicine, v. 235, n. 4, p. 497-505, APR 2010. Web of Science Citations: 37. (09/03143-1, 06/61523-7)
BUDAS, GRANT; COSTA, JR., HELIO MIRANDA; BATISTA FERREIRA, JULIO CESAR; DA SILVA FERREIRA, ANDRE TEIXEIRA; PERALES, JONAS; KRIEGER, JOSE EDUARDO; MOCHLY-ROSEN, DARIA; SCHECHTMAN, DEBORAH. Identification of epsilon PKC Targets During Cardiac Ischemic Injury. CIRCULATION JOURNAL, v. 76, n. 6, p. 1476-1485, JUN 2012. Web of Science Citations: 22. (05/54188-4, 09/03143-1)
FERREIRA, JULIO C. B.; BOER, BERTA NAPCHAN; GRINBERG, MAX; BRUM, PATRICIA CHAKUR; MOCHLY-ROSEN, DARIA. Protein Quality Control Disruption by PKC beta II in Heart Failure; Rescue by the Selective PKC beta II Inhibitor, beta IIV5-3. PLoS One, v. 7, n. 3, MAR 30 2012. Web of Science Citations: 31. (09/03143-1)
UETA, CINTIA BAGNE; CAMPOS, JULIANE CRUZ; PRESTES E ALBUQUERQUE, RUDA; LIMA, VANESSA MORAIS; DISATNIK, MARIE-HELENE; SANCHEZ, ANGELICA BIANCHINI; CHEN, CHE-HONG; GENNARI DE MEDEIROS, MARISA HELENA; YANG, WENJIN; MOCHLY-ROSEN, DARIA; et al. Cardioprotection induced by a brief exposure to acetaldehyde: role of aldehyde dehydrogenase 2. Cardiovascular Research, v. 114, n. 7, p. 1006-1015, JUN 1 2018. Web of Science Citations: 10. (15/20783-5, 13/11315-2, 12/05765-2, 13/07937-8)
BATISTA FERREIRA, JULIO CESAR; BRUM, PATRICIA CHAKUR; MOCHLY-ROSEN, DARIA. beta IIPKC and epsilon PKC isozymes as potential pharmacological targets in cardiac hypertrophy and heart failure. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, v. 51, n. 4, p. 479-484, OCT 2011. Web of Science Citations: 31. (09/03143-1)
DISATNIK, MARIE-HELENE; FERREIRA, JULIO C. B.; CAMPOS, JULIANE CRUZ; GOMES, KATIA SAMPAIO; DOURADO, PAULO M. M.; QI, XIN; MOCHLY-ROSEN, DARIA. Acute Inhibition of Excessive Mitochondrial Fission After Myocardial Infarction Prevents Long-term Cardiac Dysfunction. JOURNAL OF THE AMERICAN HEART ASSOCIATION, v. 2, n. 5, OCT 2013. Web of Science Citations: 105. (12/05765-2)
CHEN, CHE-HONG; BATISTA FERREIRA, JULIO CESAR; GROSS, ERIC R.; MOCHLY-ROSEN, DARIA. TARGETING ALDEHYDE DEHYDROGENASE 2: NEW THERAPEUTIC OPPORTUNITIES. Physiological Reviews, v. 94, n. 1, p. 1-34, JAN 2014. Web of Science Citations: 160. (12/05765-2)
BOZI, LUIZ H. M.; CAMPOS, JULIANE C.; ZAMBELLI, VANESSA O.; FERREIRA, NIKOLAS D.; FERREIRA, JULIO C. B.. Mitochondrially-targeted treatment strategies. MOLECULAR ASPECTS OF MEDICINE, v. 71, FEB 2020. Web of Science Citations: 0. (13/07937-8, 17/16071-5, 18/18627-3, 17/24836-1, 17/16540-5, 17/16694-2, 18/19332-7)
TAKANO, ANA P. C.; CAMPOS, JULIANE C.; VOLTARELLI, VANESSA A.; FERREIRA, JULIO C. B.. Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3. INTERNATIONAL JOURNAL OF CARDIOLOGY, v. 272, p. 194-201, DEC 1 2018. Web of Science Citations: 2. (16/09611-0, 15/22814-5)
ALVES, CHRISTIANO R. R.; FARIA, DANIELE DE P.; CARNEIRO, CAMILA DE G.; GARCEZ, ALEXANDRE T.; GUTIERREZ, VANESSA P.; DAS NEVES, WILLIAN; DE ALMEIDA, NEY R.; CURY, YARA; CHAMMAS, ROGER; BRUM, PATRICIA C.. F-18-Fluoride PET/CT and Tc-99m-MDP SPECT/CT can detect bone cancer at early stage in rodents. Life Sciences, v. 206, p. 29-34, AUG 1 2018. Web of Science Citations: 0. (15/22814-5, 14/03016-8, 16/01478-0, 12/02528-0, 12/25240-1)
UETA, CINTIA BAGNE; CAMPOS, JULIANE CRUZ; PRESTES E ALBUQUERQUE, RUDA; LIMA, VANESSA MORAIS; DISATNIK, MARIE-HELENE; SANCHEZ, ANGELICA BIANCHINI; CHEN, CHE-HONG; GENNARI DE MEDEIROS, MARISA HELENA; YANG, WENJIN; MOCHLY-ROSEN, DARIA; et al. Cardioprotection induced by a brief exposure to acetaldehyde: role of aldehyde dehydrogenase 2. Cardiovascular Research, v. 114, n. 7, p. 1006-1015, JUN 1 2018. Web of Science Citations: 10. (15/20783-5, 13/11315-2, 12/05765-2, 13/07937-8)
GOMES, KATIA M. S.; CAMPOS, JULIANE C.; BECHARA, LUIZ R. G.; QUELICONI, BRUNO; LIMA, VANESSA M.; DISATNIK, MARIE-HELENE; MAGNO, PAULO; CHEN, CHE-HONG; BRUM, PATRICIA C.; KOWALTOWSKI, ALICIA J.; et al. Aldehyde dehydrogenase 2 activation in heart failure restores mitochondrial function and improves ventricular function and remodelling. Cardiovascular Research, v. 103, n. 4, p. 498-508, SEP 1 2014. Web of Science Citations: 54. (12/05765-2)
FERREIRA, JULIO C. B.; MOREIRA, JOSE B. N.; CAMPOS, JULIANE C.; PEREIRA, MARCELO G.; MATTOS, KATT C.; COELHO, MARCELE A.; BRUM, PATRICIA C.. Angiotensin receptor blockade improves the net balance of cardiac Ca2+ handling-related proteins in sympathetic hyperactivity-induced heart failure. Life Sciences, v. 88, n. 13-14, p. 578-585, MAR 28 2011. Web of Science Citations: 17. (09/03143-1, 05/59740-7)
FERREIRA, JULIO C. B.; MOCHLY-ROSEN, DARIA. Nitroglycerin Use in Myocardial Infarction Patients - Risks and Benefits. CIRCULATION JOURNAL, v. 76, n. 1, p. 15-21, JAN 2012. Web of Science Citations: 32. (09/03143-1)
GOMES, KATIA M. S.; CAMPOS, JULIANE C.; BECHARA, LUIZ R. G.; QUELICONI, BRUNO; LIMA, VANESSA M.; DISATNIK, MARIE-HELENE; MAGNO, PAULO; CHEN, CHE-HONG; BRUM, PATRICIA C.; KOWALTOWSKI, ALICIA J.; et al. Aldehyde dehydrogenase 2 activation in heart failure restores mitochondrial function and improves ventricular function and remodelling. Cardiovascular Research, v. 103, n. 4, p. 498-508, SEP 1 2014. Web of Science Citations: 54. (12/05765-2)
CAMPOS, JULIANE C.; GOMES, KATIA M. S.; FERREIRA, JULIO C. B.. Impact of exercise training on redox signaling in cardiovascular diseases. Food and Chemical Toxicology, v. 62, p. 107-119, DEC 2013. Web of Science Citations: 36. (12/05765-2, 12/14416-1)
UETA, CINTIA B.; GOMES, KATIA S.; RIBEIRO, MARCIO A.; MOCHLY-ROSEN, DANIA; FERREIRA, JULIO C. B.. Disruption of mitochondrial quality control in peripheral artery disease: New therapeutic opportunities. PHARMACOLOGICAL RESEARCH, v. 115, p. 96-106, JAN 2017. Web of Science Citations: 8. (15/20783-5, 13/24321-0, 15/01759-6, 12/05765-2, 14/15187-1)
GOMES, KATIA M. S.; BECHARA, LUIZ R. G.; LIMA, VANESSA M.; RIBEIRO, MARCIO A. C.; CAMPOS, JULIANE C.; DOURADO, PAULO M.; KOWALTOWSKI, ALICIA J.; MOCHLY-ROSEN, DARIA; FERREIRA, JULIO C. B.. Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: Benefits of Alda-1. INTERNATIONAL JOURNAL OF CARDIOLOGY, v. 179, p. 129-138, JAN 20 2015. Web of Science Citations: 26. (12/05765-2, 10/51906-1, 13/07937-8)
RAMESH, MRIDULA; CAMPOS, JULIANE C.; LEE, PAMELA; SONG, YANG; HERNANDEZ, GENARO; SIN, JON; TUCKER, KYLE C.; SAADAEIJAHROMI, HANNANEH; GURNEY, MICHAEL; FERREIRA, JULIO C. B.; et al. Mitophagy protects against statin-mediated skeletal muscle toxicity. FASEB JOURNAL, v. 33, n. 11, p. 11857-11869, NOV 2019. Web of Science Citations: 0. (16/01633-5)
CHEN, CHE-HONG; FERREIRA, JULIO C. B.; JOSHI, AMIT U.; STEVENS, MATTHEW C.; LI, SIN-JIN; HSU, JADE H. -M.; MACLEAN, RORY; FERREIRA, NIKOLAS D.; CERVANTES, PILAR R.; MARTINEZ, DIANA D.; et al. Novel and prevalent non-East Asian ALDH2 variants; Implications for global susceptibility to aldehydes' toxicity. EBIOMEDICINE, v. 55, MAY 2020. Web of Science Citations: 0. (18/18627-3)
CAMPOS, JULIANE C.; CHEN, CHE-HONG; FERREIRA, JULIO C. B.. Comment on: ``Aldehyde dehydrogenases contribute to skeletal muscle homeostasis in healthy, aging, and Duchenne muscular dystrophy patients{''} by Etienne et al.. JOURNAL OF CACHEXIA SARCOPENIA AND MUSCLE, JUL 2020. Web of Science Citations: 0. (15/22814-5, 17/16540-5, 19/25049-9, 19/18444-9)
CAMPOS, JULIANE C.; BAEHR, LESLIE M.; FERREIRA, NIKOLAS D.; BOZI, LUIZ H. M.; ANDRES, ALLEN M.; RIBEIRO, MARCIO A. C.; GOTTLIEB, ROBERTA A.; BODINE, SUE C.; FERREIRA, JULIO C. B.. beta(2)-adrenoceptor activation improves skeletal muscle autophagy in neurogenic myopathy. FASEB JOURNAL, v. 34, n. 4, FEB 2020. Web of Science Citations: 0. (18/18627-3, 16/01633-5, 17/16540-5, 12/14416-1, 15/50429-9, 17/16694-2)
CAMPOS, JULIANE C.; QUELICONI, BRUNO B.; DOURADO, PAULO M. M.; CUNHA, TELMA F.; ZAMBELLI, VANESSA O.; BECHARA, LUIZ R. G.; KOWALTOWSKI, ALICIA J.; BRUM, PATRICIA C.; MOCHLY-ROSEN, DARIA; FERREIRA, JULIO C. B.. Exercise Training Restores Cardiac Protein Quality Control in Heart Failure. PLoS One, v. 7, n. 12, DEC 27 2012. Web of Science Citations: 44. (12/05765-2, 10/00028-4, 09/12349-2, 09/18546-4)
FERREIRA, JULIO C. B.; KOYANAGI, TOMOYOSHI; PALANIYANDI, SURESH S.; FAJARDO, GIOVANNI; CHURCHILL, ERIC N.; BUDAS, GRANT; DISATNIK, MARIE-HELENE; BERNSTEIN, DANIEL; BRUM, PATRICIA C.; MOCHLY-ROSEN, DARIA. Pharmacological inhibition of beta IIPKC is cardioprotective in late-stage hypertrophy. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, v. 51, n. 6, p. 980-987, DEC 2011. Web of Science Citations: 24. (09/03143-1)
CAMPOS, JULIANE C.; QUELICONI, BRUNO B.; BOZI, LUIZ H. M.; BECHARA, LUIZ R. G.; DOURADO, PAULO M. M.; ANDRES, ALLEN M.; JANNIG, PAULO R.; GOMES, KATIA M. S.; ZAMBELLI, VANESSA O.; ROCHA-RESENDE, CIBELE; et al. Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure. AUTOPHAGY, v. 13, n. 8, p. 1304-1317, 2017. Web of Science Citations: 18. (15/20783-5, 09/12349-2, 16/01633-5, 12/05765-2, 10/00028-4, 10/51906-1)
CAMPOS, JULIANE C.; BAEHR, LESLIE M.; GOMES, KATIA M. S.; BECHARA, LUIZ R. G.; VOLTARELLI, VANESSA A.; BOZI, LUIZ H. M.; RIBEIRO, MARCIO A. C.; FERREIRA, NIKOLAS D.; MOREIRA, JOSE B. N.; BRUM, PATRICIA C.; et al. Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy. SCIENTIFIC REPORTS, v. 8, AUG 7 2018. Web of Science Citations: 4. (15/20783-5, 15/22814-5, 12/14416-1, 16/01633-5)
CUNHA, TELMA F.; BECHARA, LUIZ R. G.; BACURAU, ALINE V. N.; JANNIG, PAULO R.; VOLTARELLI, VANESSA A.; DOURADO, PAULO M.; VASCONCELOS, ANDREA R.; SCAVONE, CRISTOFORO; FERREIRA, JULIO C. B.; BRUM, PATRICIA C.. Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats. Journal of Applied Physiology, v. 122, n. 4, p. 817-827, APR 2017. Web of Science Citations: 8. (12/05765-2, 11/00728-9, 14/23703-0)
CAMPOS, JULIANE C.; BOZI, LUIZ H. M.; BECHARA, LUIZ R. G.; LIMA, VANESSA M.; FERREIRA, JULIO C. B.. Mitochondrial Quality Control in Cardiac Diseases. FRONTIERS IN PHYSIOLOGY, v. 7, OCT 21 2016. Web of Science Citations: 12. (15/20783-5, 12/14416-1, 16/01633-5, 12/05765-2, 12/19379-7)
FERREIRA, JULIO C. B.; BOER, BERTA NAPCHAN; GRINBERG, MAX; BRUM, PATRICIA CHAKUR; MOCHLY-ROSEN, DARIA. Protein Quality Control Disruption by PKC beta II in Heart Failure; Rescue by the Selective PKC beta II Inhibitor, beta IIV5-3. PLoS One, v. 7, n. 3, MAR 30 2012. Web of Science Citations: 31. (09/03143-1)
BUDAS, GRANT; COSTA, JR., HELIO MIRANDA; BATISTA FERREIRA, JULIO CESAR; DA SILVA FERREIRA, ANDRE TEIXEIRA; PERALES, JONAS; KRIEGER, JOSE EDUARDO; MOCHLY-ROSEN, DARIA; SCHECHTMAN, DEBORAH. Identification of epsilon PKC Targets During Cardiac Ischemic Injury. CIRCULATION JOURNAL, v. 76, n. 6, p. 1476-1485, JUN 2012. Web of Science Citations: 22. (05/54188-4, 09/03143-1)
SOUZA, RODRIGO W. A.; ALVES, CHRISTIANO R. R.; MEDEIROS, ALESSANDRA; ROLIM, NATALE; SILVA, GUSTAVO J. J.; MOREIRA, JOSE B. N.; ALVES, MARCIA N.; WOHLWEND, MARTIN; GEBRIEL, MOHAMMED; HAGEN, LARS; et al. Differential regulation of cysteine oxidative post-translational modifications in high and low aerobic capacity. SCIENTIFIC REPORTS, v. 8, DEC 11 2018. Web of Science Citations: 1. (14/03016-8, 14/25957-9, 12/25240-1, 15/22814-5)
FERREIRA, JULIO C. B.; CAMPOS, JULIANE C.; QVIT, NIR; QI, XIN; BOZI, LUIZ H. M.; BECHARA, LUIZ R. G.; LIMA, VANESSA M.; QUELICONI, BRUNO B.; DISATNIK, MARIE-HELENE; DOURADO, PAULO M. M.; et al. A selective inhibitor of mitofusin 1-beta IIPKC association improves heart failure outcome in rats. NATURE COMMUNICATIONS, v. 10, JAN 18 2019. Web of Science Citations: 4. (17/16694-2, 15/20783-5, 16/09611-0, 15/22814-5, 17/11142-1, 09/12349-2, 16/01633-5, 12/05765-2, 10/00028-4, 17/16540-5, 10/51906-1)
RAMESH, MRIDULA; CAMPOS, JULIANE C.; LEE, PAMELA; SONG, YANG; HERNANDEZ, GENARO; SIN, JON; TUCKER, KYLE C.; SAADAEIJAHROMI, HANNANEH; GURNEY, MICHAEL; FERREIRA, JULIO C. B.; et al. Mitophagy protects against statin-mediated skeletal muscle toxicity. FASEB JOURNAL, v. 33, n. 11, p. 11857-11869, NOV 2019. Web of Science Citations: 0. (16/01633-5)
CHEN, CHE-HONG; FERREIRA, JULIO C. B.; JOSHI, AMIT U.; STEVENS, MATTHEW C.; LI, SIN-JIN; HSU, JADE H. -M.; MACLEAN, RORY; FERREIRA, NIKOLAS D.; CERVANTES, PILAR R.; MARTINEZ, DIANA D.; et al. Novel and prevalent non-East Asian ALDH2 variants; Implications for global susceptibility to aldehydes' toxicity. EBIOMEDICINE, v. 55, MAY 2020. Web of Science Citations: 0. (18/18627-3)
CAMPOS, JULIANE C.; BAEHR, LESLIE M.; FERREIRA, NIKOLAS D.; BOZI, LUIZ H. M.; ANDRES, ALLEN M.; RIBEIRO, MARCIO A. C.; GOTTLIEB, ROBERTA A.; BODINE, SUE C.; FERREIRA, JULIO C. B.. beta(2)-adrenoceptor activation improves skeletal muscle autophagy in neurogenic myopathy. FASEB JOURNAL, v. 34, n. 4, FEB 2020. Web of Science Citations: 0. (18/18627-3, 16/01633-5, 17/16540-5, 12/14416-1, 15/50429-9, 17/16694-2)
CAMPOS, JULIANE C.; CHEN, CHE-HONG; FERREIRA, JULIO C. B.. Comment on: ``Aldehyde dehydrogenases contribute to skeletal muscle homeostasis in healthy, aging, and Duchenne muscular dystrophy patients{''} by Etienne et al.. JOURNAL OF CACHEXIA SARCOPENIA AND MUSCLE, JUL 2020. Web of Science Citations: 0. (15/22814-5, 17/16540-5, 19/25049-9, 19/18444-9)
(References retrieved automatically from State of São Paulo Research Institutions)
ANDRADE, Kátia Maria Gomes. Participação da enzima aldeído desidrogenase 2 na insuficiência cardíaca induzida por infarto do miocárdio.. Dissertação (Mestrado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (12/02654-5)
KIYUNA, Ligia Akemi. Análise da atividade e expressão da proteína Dicer em condições de estresse de aldeídos: possível papel protetor da enzima ALDH2.. Dissertação (Mestrado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (16/00900-0)
RIBEIRO, Márcio Augusto Campos. Participação da enzima aldeído desidrogenase 2 na progressão da doença arterial periférica.. Dissertação (Mestrado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (15/01759-6)
CAMPOS, Juliane Cruz. Controle de qualidade de proteína na disfunção/atrofia muscular esquelética: papel do receptor β2-adrenérgico.. Tese (Doutorado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (12/14416-1)
DOMINGUES, Laís Santos. Validação da atividade do eixo intracelular PKCépsilon-ALDH2 como mecanismo-chave na cardioproteção induzida pelo exercício físico.. Dissertação (Mestrado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (15/04462-4)
LIMA, Vanessa Morais. Papel da mutação ALDH2*2 na insuficiência cardíaca: potencial terapêutico da Alda-1.. Tese (Doutorado) - Instituto de Ciências Biomédicas. Universidade de São Paulo (USP). São Paulo. (12/19379-7)
FERREIRA, Julio Cesar Batista. Participação da isoforma proteína quinase C beta II na insuficiência cardíaca. xvi, 92 f. Tese (Doutorado) - Escola de Educação Física e Esporte. Universidade de São Paulo (USP). São Paulo. (06/56321-6)