I am a Postdoctoral Researcher at the Department of Pharmacology, Ribeirão Preto Medical School, University of São Paulo. With a solid academic foundation, I hold a PhD in Biological Sciences (Pharmacology) from the same institution, including international experience during a sandwich period at Universität Regensburg (Germany). My educational background also includes a Master's degree in Pharmaceutical Sciences and a Bachelor's degree in Pharmacy, both from the Federal University of Pará, where I also earned certifications in Biochemistry and Pharmacology. My research expertise lies in preclinical studies using rodent models to investigate the mechanisms underlying stress-related diseases, particularly the interplay between depression and cardiovascular disorders. I have extensive experience in designing and conducting experiments, data analysis, and employing molecular and behavioral approaches to address key questions in translational pharmacology. Passionate about advancing knowledge in the fields of neuropsychopharmacology and cardiovascular research, I aim to contribute to innovative strategies for understanding and treating complex disorders. (Source: Lattes Curriculum)
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Studies using models of social stressors are increasingly important, as they are related to pathological processes. The social defeat model promotes defensive behaviors in rats, especially passive cope, in submissive animals. Repeated or excessive exposure to social stress promotes behavioral (depressive and anhedonic-like behaviors), cardiovascular (increased heart rate) and dysautonomia…
Stress is an organism's condition in response to exposure to adverse environmental situations, involving essential adaptive responses for survival. The activation of the hypothalamus adrenal-axis, autonomic nervous system and brain structures (eg.: brainstem and amygdala), have an important role in stress, and consequently also in the related pathophysiological process. Oxytocin can be a …
Stress is a situation common in life and demands physiological and behavioral changes to reestablish homeostatic equilibrium. Stressor exposure may cause stressor-dependent responses including behavioral and neuroendocrine responses as activation of the HPA axis and the oxytocin (OXT) system. OXT secretion into the blood has been observed in response to acute psychological and social stre…
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