CV Lattes
ResearcherID
ORCID
Universidade de São Paulo (USP). Faculdade de Medicina de Ribeirão Preto (FMRP) (Institutional affiliation from the last research proposal) Birthplace: Brazil
graduate at Biologia from Centro Universitário Barão de Mauá (2000), master's at Análises Clínicas from Faculdade de Ciências Farmacêuticas de Ribeirão Preto (2004) and ph.d. at Sanduíche (Experiência Internacional) from Centre National de la Recherche Scientifique (2007). Has experience in Immunology, focusing on Celular Imunology, acting on the following subjects: mycobacterium tuberculosis, mast cells, toxocara canis, histamine and tuberculosis. (Source: Lattes Curriculum)
| Articles published in Pesquisa FAPESP Magazine about the researcher: |
| Pistas del origen de la diabetes tipo 1 |
| Clues about the origin of type 1 diabetes |
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| In Situ Cell Therapy wins eAwards Brazil 2023 |
| Biodressing accelerates skin wound healing in diabetic mice |
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| News published in Agência FAPESP Newsletter about the researcher |
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| Articles published in other media outlets ( ): |
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| VEICULO: TITULO (DATA) |
| VEICULO: TITULO (DATA) |
Type 2 diabetes (T2D) and obesity represent growing public health challenges, strongly associated with insulin resistance. Recent studies point to the gut microbiota as a determining factor in this process, as intestinal dysbiosis can promote metainflammation in visceral adipose tissue (VAT), intestinal permeability, and, consequently, the development of T2D. In this scenario, prebiotics …
The etiology of DM is multifactorial related to genetic, environmental, food and metabolic factors. Several findings reveal an association between diet, gut Dysbiosis and the activation of immunological mechanisms, which results in the pathophysiology of type 1 and type 2 Diabetes Mellitus (T1D and T2D). It is shown that probiotics control the gut Dysbiosis, improve the intestinal permeab…
Although a correlation between polymorphisms of NOD-like receptor family-pyrin domain containing 3 (NLRP3) and predisposition to type 1 diabetes (T1D) has been identified, the potential function and activation of the NLRP3 inflammasome in T1D have not been clarified. The present study shows that non-obese diabetic (NOD) mice exhibited increased NLRP3, apoptosis-associated speck-like prot…
Alarming rates of diabetes mellitus (DM) demonstrated that this disease has become a major problem in public health field worldwide. Currently, there are about six million of diabetic patients in Brazil. The etiology of diabetes remains unclear, but it is known that the risk of developing the disease is determined by genetic factors and the interference of environmental factors including …
Diabetes mellitus (DM) is a metabolic disorder characterized by persistent hyperglycemia due to dysfunction in insulin production or action. Type 1 diabetes (T1D), an autoimmune form of the disease, results from the destruction of pancreatic beta cells due to an autoimmune response mediated by autoreactive T and B lymphocytes. Several studies have demonstrated that intestinal dysbiosis an…
The SARS-CoV-2 virus rapidly spread worldwide after the identification of the first case in Wuhan, and the disease continues to impact public health and attract the attention of researchers. One of the main problems of the COVID-19 pandemic is the diversity of symptoms and the varying clinical forms among patients. Additionally, many patients develop long-term complications known as long …
(Only some records are available in English at this moment)
Type 1 diabetes is a metabolic disease of a multisystemic nature, notorious for its clinical presentation characterized by hyperglycemia and propensity to ketoacidosis. This condition results from insulin deficiency, a hormone whose production is compromised due to the response by T cells. In this condition, the organism produces autoantibodies directed to pancreatic beta cells, located i…
(Only some records are available in English at this moment)
Obesity, a chronic condition with multiple causes, predisposes individuals to metabolic syndrome (MetS) and type 2 diabetes (T2D) by disrupting energy balance through lipid accumulation and the infiltration of inflammatory cells into adipose tissue (AT) named meta-inflammation. A high-fat diet (HFD) is linked to decreased levels of intestinal Th17 cells and segmented filamentous bacteria …