CV Lattes
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Universidade São Francisco (USF). Campus Bragança Paulista (Institutional affiliation from the last research proposal) Birthplace: Brazil
graduate at Ciências Biológicas from Pontifícia Universidade Católica de Campinas (1998), master's at Clinical Medicine from Universidade Estadual de Campinas (2001) and ph.d. at Pharmacology from Universidade Estadual de Campinas (2004). Has experience in Genetics, focusing on Molecular Genetics and of Microorganisms, acting on the following subjects: helicobacter pylori, gastric cancer and DNA repair; obesity and adipogenesis (Source: Lattes Curriculum)
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Gastric cancer remains one of the leading causes of global cancer mortality. It has been shown that gastric cancer may originate from adult gastric stem cells and that it contains a subpopulation of cancer cells with stem cell characteristics, which are linked to Helicobacter pylori infection, therapy resistance and metastasis. Thus, the identification of transcription factors and related…
It is known that H. pylori infection triggers a pathological progression in the gastric mucosa which eventually culminates in the development of gastric cancer. Inevitably, the infection causes an inflammatory response in the host, which generates reactive oxygen species and nitrogen, which can cause protein alterations, damage to cell membranes and oxidation of the DNA molecule, which ar…
It has been hypothesised that genetic variation in base excision repair (BER) might modify colorectal adenoma risk. Thus, we evaluated the influence of APE1 T2197G (Asp148Glu) polymorphism on APE1, XRCC1, PARP1, and OGG1 expression in normal and tumour samples from patients with colorectal cancer. The results indicate a down-regulation of OGG1 and an up-regulation of XRCC1 expression in t…
(Only some records are available in English at this moment)
It is known that H. pylori infection triggers a pathological progression in gastric mucosa, which eventually culminates in the development of gastric cancer. Inevitably, the infection causes an inflammatory response in the host, which generates reactive oxygen species and nitrogen, which may cause protein alterations, damage to cell membranes and oxidation DNA molecule, which are known to…
It is known that H. pylori infection triggers a pathological progression in gastric mucosa which eventually culminates in the development of gastric cancer. Inevitably, the infection causes an inflammatory response in the host, which generates reactive species of oxygen and nitrogen, which can cause protein alterations, damage to cell membranes and oxidation to the DNA molecule, which are…
It is now known that gastric cancer is primarily a disease with an epigenetic origin. It has been shown that infection by H. pylori triggers a pathological progression in gastric mucosa that starts with chronic gastritis progresses to atrophic gastritis, intestinal metaplasia, dysplasia and eventually gastric cancer. It is believed that the inflammatory response generated by the infection…
(Only some records are available in English at this moment)
| 11 / 9 | Completed research grants |
| 16 / 9 | Completed scholarships in Brazil |
| 27 / 18 | All research grants and scholarships |
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